Changes in sphingomyelinases, ceramide, Bax, Bcl2, and caspase-3 during and after experimental status epilepticus

Epilepsy Research

Volumn 81, Issue 2-3, 2008, Pages 161-166

  Mikati, Mohamad A ^a   Zeinieh, Michele ^a   Habib, Ralph Abi ^a   El Hokayem, Jimmy ^a   Rahmeh, Amal ^a   El Sabban, Marwan ^a   Usta, Julnar ^a   Dbaibo, Ghassan ^a  

^a AMERICAN UNIVERSITY OF BEIRUT   (Lebanon)

Author keywords

Bax; Caspase 3; Ceramide; Kainic acid; Sphingomyelinase; Status epilepticus

Indexed keywords

CASPASE 3; CERAMIDE; KAINIC ACID; PROTEIN BAX; PROTEIN BCL 2; SPHINGOMYELIN; SPHINGOMYELIN PHOSPHODIESTERASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; EPILEPTIC STATE; IMMUNOHISTOCHEMISTRY; INJECTION; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; RAT;

ANIMALS; ANTICONVULSANTS; BCL-2-ASSOCIATED X PROTEIN; CASPASE 3; CELL DEATH; CERAMIDES; DIAZEPAM; DISEASE MODELS, ANIMAL; GENE EXPRESSION REGULATION; IN SITU NICK-END LABELING; KAINIC ACID; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; RATS, SPRAGUE-DAWLEY; SPHINGOMYELIN PHOSPHODIESTERASE; STATUS EPILEPTICUS; TIME FACTORS;

EID: 53249156006     PISSN: 09201211     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.eplepsyres.2008.05.009     Document Type: Article

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