Status epilepticus-induced neuronal damage in the rat amygdaloid complex: distribution, time-course and mechanisms

Neuroscience

Volumn 94, Issue 2, 1999, Pages 473-495

  Tuunanen, J ^a   Lukasiuk, K ^a   Halonen, T ^a   Pitkanen A ^a,b  

^a UNIVERSITY OF EASTERN FINLAND   (Finland)

^b KUOPIO UNIVERSITY HOSPITAL   (Finland)

Author keywords

Apoptosis; Bax; Bcl 2; Epilepsy; Kainate; TUNEL

Indexed keywords

KAINIC ACID; PROTEIN BAX; PROTEIN BCL 2; TRANSFERASE;

AMYGDALOID NUCLEUS; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CONTROLLED STUDY; DISEASE ACTIVITY; ELECTROCORTICOGRAPHY; EPILEPTIC DISCHARGE; EPILEPTIC STATE; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; IMMUNOREACTIVITY; MALE; NERVE INJURY; NONHUMAN; PRIORITY JOURNAL; RAT;

AMYGDALA; ANIMALS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; ELECTROENCEPHALOGRAPHY; KAINIC ACID; MALE; NEURONS; ORGAN SPECIFICITY; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; RATS, WISTAR; STATUS EPILEPTICUS; TIME FACTORS;

EID: 0032884344     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0306-4522(99)00251-1     Document Type: Article

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