DUSP1 is controlled by p53 during the cellular response to oxidative stress

Molecular Cancer Research

Volumn 6, Issue 4, 2008, Pages 624-633

  Liu, Yu Xin ^a   Wang, Jianli ^a,c   Guo, Jianfen ^a   Wu, Jingjing ^a   Lieberman, Howard B ^a,b   Yin, Yuxin ^a,b  

^a College of Physicians and Surgeons ^*  (United States)

^b Columbia University ^*  (United States)

^c MEDICAL SCHOOL OF XI AN JIAOTONG UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

DUSP1 PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN P53; PROTEIN TYROSINE PHOSPHATASE; UNCLASSIFIED DRUG;

ANIMAL CELL; APOPTOSIS; ARTICLE; CANCER CELL; CELL DEATH; COLON CANCER; CONTROLLED STUDY; DUSP1 GENE; EMBRYO; ENZYME ACTIVITY; ENZYME INHIBITION; ENZYME SPECIFICITY; FIBROBLAST; GENE; GENE OVEREXPRESSION; HUMAN; HUMAN CELL; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DEPHOSPHORYLATION;

ANIMALS; APOPTOSIS; BASE SEQUENCE; CELL LINE, TUMOR; CHROMATIN IMMUNOPRECIPITATION; DUAL SPECIFICITY PHOSPHATASE 1; HUMANS; MICE; MITOGEN-ACTIVATED PROTEIN KINASES; MOLECULAR SEQUENCE DATA; OXIDATIVE STRESS; PROMOTER REGIONS (GENETICS); PROTEIN BINDING; SIGNAL TRANSDUCTION; TRANS-ACTIVATION (GENETICS); TRANSCRIPTION, GENETIC; TUMOR SUPPRESSOR PROTEIN P53;

EID: 42049106601     PISSN: 15417786     EISSN: None     Source Type: Journal    
DOI: 10.1158/1541-7786.MCR-07-2019     Document Type: Article

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