Anisomycin abrogates repression of protooncogene c-fos transcription in E1A + cHa-ras-transformed cells through activation of MEK/ERK kinase cascade

Journal of Cellular Biochemistry

Volumn 103, Issue 3, 2008, Pages 1005-1012

  Kukushkin, Alexander N ^a   Svetlikova, Svetlana B ^a   Amanzholov, Rustam A ^a   Pospelov, Valery A ^a  

^a INSTITUTE OF CYTOLOGY   (Russian Federation)

Author keywords

Anisomycin; E1A and cHa ras oncogenes; MEK ERK kinases; Protooncogene c fos

Indexed keywords

ACETYLCYSTEINE; ANISOMYCIN; HISTONE; HISTONE DEACETYLASE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE P38; PHOSPHATASE; PROTEIN MKP; REACTIVE OXYGEN METABOLITE; STRESS ACTIVATED PROTEIN KINASE; TRANSCRIPTION FACTOR ELK 1; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; CELL LINE; CONTROLLED STUDY; DRUG MECHANISM; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME ASSAY; ENZYME PHOSPHORYLATION; FIBROBLAST; GENE; GENE REPRESSION; GENETIC TRANSCRIPTION; IEG GENE; IN VITRO STUDY; NONHUMAN; NUCLEOSOME; ONCOGENE C FOS; ONCOGENE C H RAS; ONCOGENE E1A; ONCOGENE RAS; PRIORITY JOURNAL; PROTO ONCOGENE; RAT; TRANSCRIPTION INITIATION;

ADENOVIRUS E1A PROTEINS; ANIMALS; ANISOMYCIN; CELL LINE, TRANSFORMED; CELL TRANSFORMATION, NEOPLASTIC; ENZYME ACTIVATION; ETS-DOMAIN PROTEIN ELK-1; GENE EXPRESSION REGULATION, NEOPLASTIC; GENE SILENCING; GENES, FOS; GENES, RAS; IMMEDIATE-EARLY PROTEINS; MAP KINASE SIGNALING SYSTEM; MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-FOS; RATS; TRANS-ACTIVATION (GENETICS);

RATTUS;

EID: 39049085899     PISSN: 07302312     EISSN: 10974644     Source Type: Journal    
DOI: 10.1002/jcb.21471     Document Type: Article

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