Anisomycin uses multiple mechanisms to stimulate mitogen-activated protein kinases and gene expression and to inhibit neuronal differentiation in PC12 phaeochromocytoma cells

European Journal of Neuroscience

Volumn 12, Issue 2, 2000, Pages 527-532

  Törocsik, Beáta ^a   Szeberényi, József ^a  

^a UNIVERSITY OF PÉCS   (Hungary)

Author keywords

C Jun N terminal kinases; Extracellular signal regulated kinases; Nerve growth factor; p38 kinase; Ras proteins

Indexed keywords

ANISOMYCIN; MITOGEN ACTIVATED PROTEIN KINASE; NERVE GROWTH FACTOR; PROTEIN SYNTHESIS INHIBITOR; RAS PROTEIN; REACTIVE OXYGEN METABOLITE; STRESS ACTIVATED PROTEIN KINASE; SYNAPTOPHYSIN;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; EARLY RESPONSE GENE; ENZYME ACTIVATION; ENZYME ACTIVITY; GENE EXPRESSION; NERVE CELL DIFFERENTIATION; NEURITE; NONHUMAN; ONCOGENE C FOS; PHEOCHROMOCYTOMA CELL; PRIORITY JOURNAL;

ANIMALS; ANISOMYCIN; CELL DIFFERENTIATION; DNA-BINDING PROTEINS; EARLY GROWTH RESPONSE PROTEIN 1; ENZYME ACTIVATION; ENZYME INHIBITORS; GENE EXPRESSION REGULATION, NEOPLASTIC; GENES, FOS; GENES, JUN; IMIDAZOLES; IMMEDIATE-EARLY PROTEINS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MACROMOLECULAR SUBSTANCES; MAP KINASE SIGNALING SYSTEM; MITOGEN-ACTIVATED PROTEIN KINASES; MODELS, BIOLOGICAL; NEOPLASM PROTEINS; NERVE GROWTH FACTOR; NEURITES; OXIDATIVE STRESS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PC12 CELLS; PROTEIN SYNTHESIS INHIBITORS; PROTO-ONCOGENE PROTEINS C-FOS; PROTO-ONCOGENE PROTEINS C-JUN; PYRIDINES; RAS PROTEINS; RATS; REACTIVE OXYGEN SPECIES; RECEPTOR, NERVE GROWTH FACTOR; RECEPTOR, TRKA; TRANSCRIPTION FACTORS;

EID: 0034050833     PISSN: 0953816X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1460-9568.2000.00933.x     Document Type: Article

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