Triadin is a critical determinant of cellular Ca cycling and contractility in the heart

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 293, Issue 5, 2007, Pages

  Kirchhefer, Uwe ^a,f   Klimas, Jan ^b   Baba, Hideo A ^c   Buchwalow, Igor B ^d   Fabritz, Larissa ^a   Hüls, Marion ^a   Matus, Marek ^a   Müller, Frank U ^a   Schmitz, Wilhelm ^a   Neumann, Joachim ^e  

^a UNIVERSITY HOSPITAL MÜNSTER   (Germany)

^b Physics and Informatics   (Slovakia)

^c UNIVERSITY HOSPITAL ESSEN   (Germany)

^d UNIVERSITY HOSPITAL MÜNSTER   (Germany)

^e MARTIN LUTHER UNIVERSITY HALLE WITTENBERG   (Germany)

^f UNIVERSITY OF MÜNSTER   (Germany)

Author keywords

adrenergic receptor stimulation; Ca signaling; Cardiac function; Force frequency relationship; Hypertrophy; Sarcoplasmic reticulum; Transgenic mice

Indexed keywords

BETA ADRENERGIC RECEPTOR; CAFFEINE; CALCIUM; ISOPRENALINE; JUNCTIN; PROTEIN; TRIADIN; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CONTROLLED STUDY; CROSS BREEDING; ECHOCARDIOGRAPHY; GENE OVEREXPRESSION; HEART FUNCTION; HEART MUSCLE CONTRACTILITY; HEART VENTRICLE HYPERTROPHY; MOUSE; NONHUMAN; PRIORITY JOURNAL; TRANSGENIC MOUSE;

ANIMALS; CALCIUM; CALCIUM SIGNALING; CARRIER PROTEINS; CELLS, CULTURED; DOGS; HEART; MICE; MICE, TRANSGENIC; MUSCLE PROTEINS; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC;

EID: 36148929388     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00799.2007     Document Type: Article

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