The commonality of protein interaction networks determined in neurodegenerative disorders (NDDs)

Bioinformatics

Volumn 23, Issue 16, 2007, Pages 2129-2138

  Limviphuvadh, Vachiranee ^a   Tanaka, Seigo ^b   Goto, Susumu ^a   Ueda, Kunihiro ^c   Kanehisa, Minoru ^a  

^a KYOTO UNIVERSITY   (Japan)

^b OSAKA OHTANI UNIVERSITY   (Japan)

^c Kobe Tokiwa Junior College   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHOTYROSINE; PROTEIN 14 3 3;

ALZHEIMER DISEASE; AMYOTROPHIC LATERAL SCLEROSIS; APOPTOSIS; ARTICLE; DEGENERATIVE DISEASE; DENTATORUBROPALLIDOLUYSIAN ATROPHY; DISEASE ASSOCIATION; GENETIC DATABASE; HUMAN; HUNTINGTON CHOREA; NONHUMAN; PARKINSON DISEASE; PATHOGENICITY; PRION DISEASE; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN DATABASE; PROTEIN DOMAIN; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION;

EID: 34548567227     PISSN: 13674803     EISSN: 13674811     Source Type: Journal    
DOI: 10.1093/bioinformatics/btm307     Document Type: Article

References (34)

1. Armstrong,R.A. et al. (2005) Overlap between neurodegenerative disorders. Neuropathology, 25, 111-124.
2. Barnham,K.J. et al. (2006) Delineating common molecular mechanisms in Alzheimer's and prion diseases. Trends Biochem. Sci., 31, 465-472.
3. Barral,J.M. et al. (2004) Roles of molecular chaperones in protein misfolding diseases. Semin. Cell Dev. Biol., 15, 17-29.
4. Borg,J.P. et al. (1996) The phosphotyrosine interaction domains of X11 and FE65 bind to distinct sites on the YENPTY motif of amyloid precursor protein. Mol. Cell Biol., 16, 6229-6241.
5. Bossy-Wetzel,E. et al. (2004) Molecular pathways to neurodegeneration. Nat. Med., 10 (Suppl.), S2-S9.
6. Bucciantini,M. et al. (2002) Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases. Nature 416, 507-511.
7. Butterfield,D.A. et al. (2006) Pin1 in Alzheimer's disease. J. Neurochem., 98, 1697-1706.
8. Caltagarone,J. et al. (2007) Focal adhesions regulate Abeta signaling and cell death in Alzheimer's disease. Biochim. Biophys. Acta, 1772, 438-445.
9. Caricasole,A. et al. (2005) Two sides of the same coin: Wnt signaling in neurodegeneration and neuro-oncology. Biosci. Rep., 25, 309-327.
10. Chuang,D.M. et al. (2005) Glyceraldehyde-3-phosphate dehydrogenase, apoptosis, and neurodegenerative diseases. Annu. Rev. Pharmacol. Toxicol., 45, 269-290.
11. Dougherty,M.K. and Morrison,D.K. (2004) Unlocking the code of 14-3-3. J. Cell Sci., 117, 1875-1884.
12. Eddy,S.R. (1998) Profile hidden Markov models. Bioinformatics, 14, 755-763.
13. Finn,R.D. et al. (2006) Pfam: Clans, web tools and services. Nucleic Acids Res., 34, D247-D251.
14. Giorgini,F. and Muchowski,P.J. (2005) Connecting the dots in Huntington's disease with protein interaction networks. Genome Biol. 6, 210.
15. Goehler,H. et al. (2004) A protein interaction network links GIT1, an enhancer of huntingtin aggregation, to Huntington's disease. Mol. Cell, 15, 853-865.
16. Haverty,P.M. et al. (2002) HugeIndex: A database with visualization tools for high-density oligonucleotide array data from normal human tissues. Nucleic Acids Res., 30, 214-217.
17. Hirokawa,N. and Takemura,R. (2004) Molecular motors in neuronal development, intracellular transport and diseases. Curr. Opin. Neurobiol., 14, 564-573.
18. Hsiao,L.L. et al. (2001) A compendium of gene expression in normal human tissues. Physiol. Genomics, 7, 97-104.
19. Kanehisa,M. et al. (2006) From genomics to chemical genomics: New developments in KEGG. Nucleic Acids Res., 34, D354-D357.
20. Kayed,R. et al. (2003) Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis. Science, 300, 486-489.
21. Kim,J.W. and Dang,C.V. (2005) Multifaceted roles of glycolytic enzymes. Trends Biochem. Sci., 30, 142-150.
22. Kudo,T. et al. (2002) The unfolded protein response is involved in the pathology of Alzheimer's disease. Ann. N. Y. Acad. Sci., 977, 349-355.
23. Macias,M.J. et al. (2002) WW and SH3 domains, two different scaffolds to recognize proline-rich ligands. FEBS Lett., 513 30-37.
24. Mackintosh,C. (2004) Dynamic interactions between 14-3-3 proteins and phosphoproteins regulate diverse cellular processes. Biochem. J., 381, 329-342.
25. Marchetti,B. et al. (2005) Glucocorticoid receptor-nitric oxide crosstalk and vulnerability to experimental parkinsonism: Pivotal role for glia-neuron interactions. Brain Res. Brain Res. Rev., 48 302-321.
26. Mathisen,P.M. (2003) Gene discovery and validation for neurodegenerative diseases. Drug Discov. Today, 8, 39-46.
27. Mazzola,J.L. and Sirover,M.A. (2002) Alteration of intracellular structure and function of glyceraldehyde-3-phosphate dehydrogenase: A common phenotype of neurodegenerative disorders? Neurotoxicology, 23, 603-609.
28. Ostrerova,N. et al. (1999) alpha-Synuclein shares physical and functional homology with 14-3-3 proteins. J. Neurosci., 19, 5782-5791.
29. Peri,S. et al. (2003) Development of human protein reference database as an initial platform for approaching systems biology in humans. Genome Res., 13, 2363-2371.
30. Rao,R.V. and Bredesen,D.E. (2004) Misfolded proteins, endoplasmic reticulum stress and neurodegeneration. Curr. Opin. Cell Biol., 16, 653-662.
31. Ross,C.A. and Poirier,M.A. (2004) Protein aggregation and neurodegenerative disease. Nat. Med., 10 (Suppl.), S10-S17.
32. Vincent,I. et al. (2003) The cell cycle and human neurodegenerative disease. Prog. Cell Cycle Res., 5, 31-41.
33. Wood,J.D. et al. (1998) Atrophin-1, the DRPLA gene product, interacts with two families of WW domain-containing proteins. Mol. Cell Neurosci., 11, 149-160.
34. Zen,K. et al. (2004) Association of BAP31 with CD11b/CD18. Potential role in intracellular trafficking of CD11b/CD18 in neutrophils. J. Biol. Chem., 279, 44924-44930.