Regulation of Bnip3 death pathways by calcium, phosphorylation, and hypoxia-reoxygenation

Antioxidants and Redox Signaling

Volumn 9, Issue 9, 2007, Pages 1309-1315

  Graham, Regina M ^a   Thompson, John W ^a   Wei, Jianqin ^a   Bishopric, Nanette H ^a   Webster, Keith A ^a  

^a UNIVERSITY OF MIAMI MILLER SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; CALCIUM IONOPHORE; DIMER; MONOMER; OKADAIC ACID; PHORBOL 13 ACETATE 12 MYRISTATE; PHOSPHATASE; PHOSPHOPROTEIN; PROTEIN BCL 2; PROTEIN BNIP3; PROTEIN KINASE C;

ACIDOSIS; AMINO ACID SEQUENCE; AMINO TERMINAL SEQUENCE; ANIMAL CELL; APOPTOSIS; ARTICLE; BNIP3 GENE; CONTROLLED STUDY; ENDOPLASMIC RETICULUM; ENZYME ACTIVATION; GENE; GENE INDUCTION; GENETIC TRANSCRIPTION; HUMAN; HUMAN TISSUE; HYPOXIA; ISCHEMIA; MITOCHONDRION; MOLECULAR SIZE; NONHUMAN; POLYACRYLAMIDE GEL ELECTROPHORESIS; PRIORITY JOURNAL; PROTEIN DEPHOSPHORYLATION; PROTEIN DOMAIN; PROTEIN FAMILY; PROTEIN INDUCTION; PROTEIN PHOSPHORYLATION; PROTEIN STABILITY; PROTEIN TRANSPORT; RAT; REGULATORY MECHANISM; REOXYGENATION; SIMULATION;

ACIDOSIS; ALKALINE PHOSPHATASE; APOPTOSIS; CALCIUM; CELL CULTURE TECHNIQUES; CELL DEATH; CELL HYPOXIA; CORONARY CIRCULATION; CORONARY VESSELS; HEART; HUMANS; MEMBRANE PROTEINS; MYOCARDIUM; OXYGEN CONSUMPTION; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS; RNA, SMALL INTERFERING;

EID: 34547875432     PISSN: 15230864     EISSN: None     Source Type: Journal    
DOI: 10.1089/ars.2007.1726     Document Type: Article

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