Physiological responses of ES-2 ovarian cell line following administration of epigallocatechin-3-gallate (EGCG), thymoquinone (TQ), and selenium (SE)

Biomedical Sciences Instrumentation

Volumn 43, Issue , 2007, Pages 378-383

  Wilson Simpson, Felisa ^a   Vance, Stacy ^a   Benghuzzi, Hamed ^a  

^a NONE

Author keywords

Antioxidants; Cell proliferation; Epigallecatechin 3 gallate (EGCG); ES 2; Estrogen; MDA; Ovarian cancer; Progesterone; Selenium; Thymoquinone

Indexed keywords

BIOMARKERS; CELL DEATH; DISEASES; DRUG PRODUCTS; METABOLISM; SELENIUM COMPOUNDS; BIOCHEMISTRY; CELL CULTURE; CELL PROLIFERATION; FREE RADICALS; HUMAN COMPUTER INTERACTION; INSTRUMENTS; LANDFORMS; PHYSIOLOGICAL MODELS; PHYSIOLOGY;

EPIGALLECATECHIN; ESTROGENS; OVARIAN CANCER; PROGESTERONE; THYMOQUINONE;

CELL PROLIFERATION; SELENIUM;

BIOCHEMICAL MARKER; EPIGALLOCATECHIN GALLATE; MALONALDEHYDE; SELENIUM; THYMOQUINONE;

ANTIOXIDANT ACTIVITY; CARCINOGENESIS; CELL COUNT; CELL LINE; CELL STRUCTURE; CONFERENCE PAPER; CONTROLLED STUDY; DRUG EFFECT; DRUG MEGADOSE; HUMAN; HUMAN CELL; LOW DRUG DOSE; METABOLISM; OVARY CANCER; OVARY CELL;

ANTINEOPLASTIC AGENTS; ANTIOXIDANTS; BENZOQUINONES; CATECHIN; CELL LINE, TUMOR; CELL PROLIFERATION; CELL SURVIVAL; DOSE-RESPONSE RELATIONSHIP, DRUG; DRUG COMBINATIONS; FEMALE; HUMANS; NEOPLASM PROTEINS; OVARIAN NEOPLASMS; SELENIUM;

ANTIOXIDANTS; ES-2; ESTROGEN; GALLATE; MDA; OVARIAN CANCERS; PROGESTERONE; THYMOQUINONE;

EID: 34247624462     PISSN: 00678856     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Conference Paper

References (20)

1. American Cancer Society. (2006). Cancer Facts and Figures. Atlanta, Ga.
2. Binn, C. W., Zhang, M. Lee, A.H., Xie, C. X. (2004). Green tea consumption enhances survival of epithelial ovarian cancer patients. Asia Pacific Journal ClinicalNutrition,13, S116.
3. Blot, W.J., Li, J.Y., Taylor, P.R., et al. (1993). Nutritional intervention trials in Linxian, China: Supplementation with specific vitamin/mineral combination, cancer incidence, and disease-specific mortality in the general population. Journal of National Cancer Institute, 85, 1483-91.
4. Fleischauer, A.T., Olson, S.H., Mignone, L., Simonsen, N., Caputo, T.A., Harlap, S. (2002). Dietary Antioxidants, Supplements, and Risk of Epithelial Ovarian Cancer. Nutritional Cancer, 40, 2, 92-98.
5. Piver, M.S., Goldberg, J.M., Tsukada, Y., et al. (1996). Characteristics of familial Ovarian cancer first 1,000 families in the Gilda Radner Familial Ovarian Cancer Registry. European Journal of Gynecology, 3, 169-76.
6. Shoieb, A.M., Elgayyar, M., Dudrick, P.S., Bell, J.L., Tithof, P.K. (2003). In vitro inhibition of growth and induction of apoptosis in cancer cell lines by thymoquinone. International Journal of Oncology, 22, 1, 107-113.
7. Yancik, R. (1993). Ovarian cancer. Age contrasts in incidence, histology, disease Stage at diagnosis of mortality. Cancer, 71, 517-23.
8. Zhang, M., Lee, A. H., Binns, C. W. (2004). Reproductive and dietary risk factors for epithelial ovarian cancer in China. Gynecological Oncology, January, 92(1), 320-326.
9. Muggia FM (2006). New and emerging intraperitonel (IP) drugs for ovarian cancer treatment. Semin Oncol Dec 33(6Suppl 12)18-24.
10. Spinella F, Roscano L, Decandia S, DiCastro V, Albini A, Elia G, Natali PG, Bagnato A (2006). Antitumor effect of green tea polyphenol epigallocatechin-3-gallate in ovarian carcinoma cells: evidence for the endothelin-1 as a potential target. Exp Biol Med Jun;231(6):1123-7.
11. Beltz LA, Bayer DK, Moss AL, Simet IM (2006). Mechanisms of cancer prevention by gren and black tea polyphenols. Anticancer Agents Med Chem Sep;6(5):389-406.
12. Tang FY, Chiang EP, Shih CJ.(2006) Green tea catechin inhibits ephrin A-1 mediated cell migration and angiogenesis of human umbilicial vein endothelial cells. J Nutr Biochem Oct [Epub ahead of print].
13. Larsson SC, Wolk A (2005). Tea consumption and ovarian cancer risk in a population based cohort. Arch Intern Med Dec 12-26; 165(22):2683-6.
14. Pan SY, Ugnat AM, Mao Y, Wen SW, Johnson KC. (2004). A case control study of diet and the risk of ovarian cancer. Cancer Epidemiol Biomarker Prev Sep; 13(9):1521-7.
15. Kantoff P.(2006). Prevention, complementary therapies, and new scientific developments in the field of prostate cancer. Rev Urol 8; Suppl 2:S9-S14.
16. Divisi D, Di Tommaso S, Salvemini S, Garramone M, Crisci R.(2006) Diet and cancer Acta Biomed Aug; 77(2): 118-23.
17. Brigelius-Flohe R, Banning A 92006). Part of the series: from dietary antioxidants to regulator in cellular signaling and gene regulation. Suloraphane and selenium, partners in adapative response and prevention of cancer. Free Radic Res. aug; 40(8):775-87.
18. Kanter M, Coskun O, Uysal H. (2006). The antioxidative and antihistamine effect of nigella sativa and its major constituent, thymoquinone on ethanol induced gastric mucosal damage. Arch Toxicol apr; 80(4):217-24.
19. Salem ML (2005). Immunomodulatory and therapeutic properties of the Nigella Sativa L. seed. Int Immunopharmacol Dec; 5(13-14):1749-70.
20. ElMahdy MA, Zhu Q, Wang QE, Wani G, Wani AA (2005). Thymoquione induces apoptosis through activation of capase-8 and mitochondrial events in p53 null myeloblastic leukemia HL-60 cells. Int J Cancer Nov 10;117(3):409-17.