Glutathione depletion in antioxidant defense of differentiated NT2-LHON cybrids

Neurobiology of Disease

Volumn 25, Issue 3, 2007, Pages 536-544

  Schoeler, S ^a   Winkler Stuck, K ^a   Szibor, R ^a   Haroon, M F ^b   Gellerich, F N ^c   Chamaon, K ^a   Mawrin, C ^a   Kirches, E ^a  

^a Department of Neuropathology ^*  (Germany)

^b OTTO VON GUERICKE UNIVERSITY   (Germany)

^c KeyNeurotek AG   (Germany)

Author keywords

Antioxidant defense; Cybrid; Glutathione; LHON; ROS

Indexed keywords

GLUTAMATE CYSTEINE LIGASE; GLUTATHIONE PEROXIDASE 1; GLUTATHIONE REDUCTASE; HYDROGEN PEROXIDE; RETINOIC ACID; SUPEROXIDE DISMUTASE;

ANTIOXIDANT ACTIVITY; ARTICLE; CELL LINE; CYBRID; GENE EXPRESSION; GENE MUTATION; GENETIC VARIABILITY; LEBER HEREDITARY OPTIC NEUROPATHY; OXIDATION REDUCTION POTENTIAL; PRIORITY JOURNAL;

ANTINEOPLASTIC AGENTS; ANTIOXIDANTS; CELL DIFFERENTIATION; CELL LINE, TUMOR; ELECTRON TRANSPORT COMPLEX I; GENOTYPE; GLUTATHIONE; GLUTATHIONE PEROXIDASE; GLUTATHIONE REDUCTASE; HUMANS; HYDROGEN PEROXIDE; MITOCHONDRIA; OPTIC ATROPHY, HEREDITARY, LEBER; OXIDANTS; POINT MUTATION; SUPEROXIDE DISMUTASE; TERATOMA; TRETINOIN;

EID: 33847239190     PISSN: 09699961     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.nbd.2006.10.014     Document Type: Article

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