Ts65Dn, a mouse model of down syndrome, exhibits increased GABA B-induced potassium current

Journal of Neurophysiology

Volumn 97, Issue 1, 2007, Pages 892-900

  Best, Tyler K ^a   Siarey, Richard J ^a   Galdzicki, Zygmunt ^a  

^a Uniformed Services University of the Health Sciences and the Walter Reed National Military Medical Center   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

4 AMINOBUTYRIC ACID B RECEPTOR; BACLOFEN; G PROTEIN COUPLED INWARDLY RECTIFYING POTASSIUM CHANNEL; G PROTEIN COUPLED INWARDLY RECTIFYING POTASSIUM CHANNEL SUBUNIT 2; POTASSIUM; PROTEIN SUBUNIT; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN; CONCENTRATION RESPONSE; CONTROLLED STUDY; DISEASE MODEL; DOWN SYNDROME; GENE OVEREXPRESSION; HIPPOCAMPUS; MOUSE; NERVE CELL CULTURE; NERVE CONDUCTION; NONHUMAN; POTASSIUM CURRENT; PRIORITY JOURNAL;

ANIMALS; ANIMALS, NEWBORN; BACLOFEN; BRAIN; CELL MEMBRANE; CELLS, CULTURED; DISEASE MODELS, ANIMAL; DOSE-RESPONSE RELATIONSHIP, DRUG; DOWN SYNDROME; EXCITATORY POSTSYNAPTIC POTENTIALS; G PROTEIN-COUPLED INWARDLY-RECTIFYING POTASSIUM CHANNELS; GABA AGONISTS; GAMMA-AMINOBUTYRIC ACID; INHIBITORY POSTSYNAPTIC POTENTIALS; MICE; MICE, NEUROLOGIC MUTANTS; NEURONS; PATCH-CLAMP TECHNIQUES; POTASSIUM; RECEPTORS, GABA-B; SYNAPTIC TRANSMISSION; TRISOMY;

EID: 33846404179     PISSN: 00223077     EISSN: 15221598     Source Type: Journal    
DOI: 10.1152/jn.00626.2006     Document Type: Article

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