Redox stress is not essential for the pseudo-hypoxic phenotype of succinate dehydrogenase deficient cells

Biochimica et Biophysica Acta - Bioenergetics

Volumn 1757, Issue 5-6, 2006, Pages 567-572

  Selak, Mary A ^a   Durán, Raul V ^a   Gottlieb, Eyal ^a  

^a BEATSON INSTITUTE FOR CANCER RESEARCH   (United Kingdom)

Author keywords

Cancer; Mitochondria; Reactive oxygen species; Succinate dehydrogenase

Indexed keywords

2 OXOGLUTARIC ACID; ANTIOXIDANT; ASCORBIC ACID; FERROUS ION; HYDROGEN PEROXIDE; HYPOXIA INDUCIBLE FACTOR 1; PROCOLLAGEN PROLINE 2 OXOGLUTARATE 4 DIOXYGENASE; SUCCINATE DEHYDROGENASE;

ARTICLE; CELL HYPOXIA; CONTROLLED STUDY; CYTOSOL; ENZYME DEFICIENCY; ENZYME INHIBITION; HUMAN; HUMAN CELL; MITOCHONDRION; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PHENOTYPE; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANTIOXIDANTS; CELL LINE; CYTOSOL; ENZYME ACTIVATION; GLUTATHIONE; HUMANS; HYDROGEN PEROXIDE; HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT; KETOGLUTARIC ACIDS; MITOCHONDRIA; MODELS, BIOLOGICAL; MUTATION; OXIDATION-REDUCTION; OXIDATIVE STRESS; OXYGEN; PHENOTYPE; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION; SUCCINATE DEHYDROGENASE; SUCCINIC ACID;

EID: 33745591371     PISSN: 00052728     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.bbabio.2006.05.015     Document Type: Article

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