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Volumn 69, Issue 4, 2006, Pages 888-898

Opposing effect of p38 MAP kinase and JNK inhibitors on the development of heart failure in the cardiomyopathic hamster

Author keywords

Cardiomyopathy; Heart failure; JNK; p38 MAP kinase

Indexed keywords

1 [7 (4 FLUOROPHENYL) 1,2,3,4 TETRAHYDRO 8 (4 PYRIDYL)PYRAZOLO[5,1 C][1,2,4]TRIAZIN 2 YL] 2 PHENYLETHANEDIONE; 4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; ANTHRA[1,9 CD]PYRAZOL 6(2H) ONE; MITOGEN ACTIVATED PROTEIN KINASE; STRESS ACTIVATED PROTEIN KINASE INHIBITOR; SYNAPTOPHYSIN;

EID: 32444446597     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cardiores.2005.11.015     Document Type: Article
Times cited : (68)

References (29)
  • 2
    • 0035834071 scopus 로고    scopus 로고
    • The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy
    • P. Liao, D. Georgakopoulos, A. Kovacs, M. Zheng, D. Lerner, and H. Pu The in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathy Proc Natl Acad Sci U S A 98 2001 12283 12288
    • (2001) Proc Natl Acad Sci U S a , vol.98 , pp. 12283-12288
    • Liao, P.1    Georgakopoulos, D.2    Kovacs, A.3    Zheng, M.4    Lerner, D.5    Pu, H.6
  • 3
    • 10044261073 scopus 로고    scopus 로고
    • P38α mitogen-activated protein kinase plays a critical role in cardiomyocyte survival but not in cardiac hypertrophic growth in response to pressure overload
    • K. Nishida, O. Yamaguchi, S. Hirotani, S. Hikoso, Y. Higuchi, and T. Watanabe p38α mitogen-activated protein kinase plays a critical role in cardiomyocyte survival but not in cardiac hypertrophic growth in response to pressure overload Mol Cell Biol 24 2004 10611 10620
    • (2004) Mol Cell Biol , vol.24 , pp. 10611-10620
    • Nishida, K.1    Yamaguchi, O.2    Hirotani, S.3    Hikoso, S.4    Higuchi, Y.5    Watanabe, T.6
  • 4
    • 0035845631 scopus 로고    scopus 로고
    • Hypertensive end-organ damage and premature mortality are p38 mitogen-activated protein kinase-dependent in a rat model of cardiac hypertrophy and dysfunction
    • T.M. Behr, S.S. Nerurkar, A.H. Nelson, R.W. Coatney, T.N. Woods, and A. Sulpizio Hypertensive end-organ damage and premature mortality are p38 mitogen-activated protein kinase-dependent in a rat model of cardiac hypertrophy and dysfunction Circulation 104 2001 1292 1298
    • (2001) Circulation , vol.104 , pp. 1292-1298
    • Behr, T.M.1    Nerurkar, S.S.2    Nelson, A.H.3    Coatney, R.W.4    Woods, T.N.5    Sulpizio, A.6
  • 5
    • 5644285411 scopus 로고    scopus 로고
    • P38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the rat
    • F. See, W. Thomas, K. Way, A. Tzanidis, A. Kompa, and D. Lewis p38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the rat J Am Coll Cardiol 44 2004 1679 1689
    • (2004) J Am Coll Cardiol , vol.44 , pp. 1679-1689
    • See, F.1    Thomas, W.2    Way, K.3    Tzanidis, A.4    Kompa, A.5    Lewis, D.6
  • 6
    • 2442575047 scopus 로고    scopus 로고
    • Targeted activation of c-Jun N-terminal kinase in vivo induces restrictive cardiomyopathy and conduction defects
    • B.G. Petrich, B.C. Eloff, D.L. Lerner, A. Kovacs, J.E. Saffitz, and D.S. Rosenbaum Targeted activation of c-Jun N-terminal kinase in vivo induces restrictive cardiomyopathy and conduction defects J Biol Chem 279 2004 15330 15338
    • (2004) J Biol Chem , vol.279 , pp. 15330-15338
    • Petrich, B.G.1    Eloff, B.C.2    Lerner, D.L.3    Kovacs, A.4    Saffitz, J.E.5    Rosenbaum, D.S.6
  • 7
    • 0036074371 scopus 로고    scopus 로고
    • The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy
    • J. Sadoshima, O. Montagne, Q. Wang, G. Wang, J. Warden, and J. Liu The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy J Clin Invest 110 2002 271 279
    • (2002) J Clin Invest , vol.110 , pp. 271-279
    • Sadoshima, J.1    Montagne, O.2    Wang, Q.3    Wang, G.4    Warden, J.5    Liu, J.6
  • 9
    • 0034829970 scopus 로고    scopus 로고
    • Stress-activated protein kinase phosphorylation during cardioprotection in the ischemic myocardium
    • R.M. Fryer, H.H. Patel, A.K. Hsu, and G.J. Gross Stress-activated protein kinase phosphorylation during cardioprotection in the ischemic myocardium Am J Physiol Heart Circ Physiol 281 2001 H1184 H1192
    • (2001) Am J Physiol Heart Circ Physiol , vol.281
    • Fryer, R.M.1    Patel, H.H.2    Hsu, A.K.3    Gross, G.J.4
  • 10
    • 0032503045 scopus 로고    scopus 로고
    • The p38-MAPK inhibitor, SB203580, inhibits cardiac stress-activated protein kinases/c-Jun N-terminal kinases (SAPKs/JNKs)
    • A. Clerk, and P.H. Sugden The p38-MAPK inhibitor, SB203580, inhibits cardiac stress-activated protein kinases/c-Jun N-terminal kinases (SAPKs/JNKs) FEBS Lett 426 1998 93 96
    • (1998) FEBS Lett , vol.426 , pp. 93-96
    • Clerk, A.1    Sugden, P.H.2
  • 11
    • 0035201877 scopus 로고    scopus 로고
    • FR167653, a p38 mitogen-activated protein kinase inhibitor, prevents Helicobacter pylori-induced gastritis in Mongolian gerbils
    • S. Takahashi, Y. Keto, T. Fujita, T. Uchiyama, and A. Yamamoto FR167653, a p38 mitogen-activated protein kinase inhibitor, prevents Helicobacter pylori-induced gastritis in Mongolian gerbils J Pharmacol Exp Ther 296 2001 48 56
    • (2001) J Pharmacol Exp Ther , vol.296 , pp. 48-56
    • Takahashi, S.1    Keto, Y.2    Fujita, T.3    Uchiyama, T.4    Yamamoto, A.5
  • 13
    • 0942298235 scopus 로고    scopus 로고
    • C-Jun is regulated by combination of enhanced expression and phosphorylation in acute-overloaded rat heart
    • W. Nadruz Jr., C.B. Kobarg, J. Kobarg, and K.G. Franchini c-Jun is regulated by combination of enhanced expression and phosphorylation in acute-overloaded rat heart Am J Physiol Heart Circ Physiol 286 2004 H760 H767
    • (2004) Am J Physiol Heart Circ Physiol , vol.286
    • Nadruz Jr., W.1    Kobarg, C.B.2    Kobarg, J.3    Franchini, K.G.4
  • 14
    • 0035143312 scopus 로고    scopus 로고
    • Protein kinase C isoform-dependent myocardial protection by ischemic preconditioning and potassium cardioplegia
    • K. Lu, H. Otani, T. Yamamura, Y. Nakao, R. Hattori, and H. Ninomiya Protein kinase C isoform-dependent myocardial protection by ischemic preconditioning and potassium cardioplegia J Thorac Cardiovasc Surg 121 2001 137 148
    • (2001) J Thorac Cardiovasc Surg , vol.121 , pp. 137-148
    • Lu, K.1    Otani, H.2    Yamamura, T.3    Nakao, Y.4    Hattori, R.5    Ninomiya, H.6
  • 15
    • 0034677931 scopus 로고    scopus 로고
    • Cleavage preferences of the apoptotic endonuclease DFF40 (caspase-activated DNase or nuclease) on naked DNA and chromatin substrates
    • P. Widlak, P. Li, X. Wang, and W.T. Garrard Cleavage preferences of the apoptotic endonuclease DFF40 (caspase-activated DNase or nuclease) on naked DNA and chromatin substrates J Biol Chem 275 2000 8226 8232
    • (2000) J Biol Chem , vol.275 , pp. 8226-8232
    • Widlak, P.1    Li, P.2    Wang, X.3    Garrard, W.T.4
  • 17
    • 0020081138 scopus 로고
    • Hereditary polymyopathy and cardiomyopathy in the Syrian hamster: I. Progression of heart and skeletal muscle lesions in the UM-X7.1 line
    • G. Jasmin, and L. Proschek Hereditary polymyopathy and cardiomyopathy in the Syrian hamster: I. Progression of heart and skeletal muscle lesions in the UM-X7.1 line Muscle Nerve 5 1982 20 25
    • (1982) Muscle Nerve , vol.5 , pp. 20-25
    • Jasmin, G.1    Proschek, L.2
  • 19
    • 11144307292 scopus 로고    scopus 로고
    • STRESS signaling pathways that modulate cardiac myocyte apoptosis
    • C.P. Baines, and J.D. Molkentin STRESS signaling pathways that modulate cardiac myocyte apoptosis J Mol Cell Cardiol 38 2005 47 62
    • (2005) J Mol Cell Cardiol , vol.38 , pp. 47-62
    • Baines, C.P.1    Molkentin, J.D.2
  • 20
    • 0037470358 scopus 로고    scopus 로고
    • Disruption of a single copy of the p38alpha MAP kinase gene leads to cardioprotection against ischemia-reperfusion
    • K. Otsu, N. Yamashita, K. Nishida, S. Hirotani, O. Yamaguchi, and T. Watanabe Disruption of a single copy of the p38alpha MAP kinase gene leads to cardioprotection against ischemia-reperfusion Biochem Biophys Res Commun 302 2003 56 60
    • (2003) Biochem Biophys Res Commun , vol.302 , pp. 56-60
    • Otsu, K.1    Yamashita, N.2    Nishida, K.3    Hirotani, S.4    Yamaguchi, O.5    Watanabe, T.6
  • 21
    • 0033748263 scopus 로고    scopus 로고
    • The role of differential activation of p38-mitogen-activated protein kinase in preconditioned ventricular myocytes
    • A.T. Saurin, J.L. Martin, R.J. Heads, C. Foley, J.W. Mockridge, and M.J. Wright The role of differential activation of p38-mitogen-activated protein kinase in preconditioned ventricular myocytes FASEB J 14 2000 2237 2246
    • (2000) FASEB J , vol.14 , pp. 2237-2246
    • Saurin, A.T.1    Martin, J.L.2    Heads, R.J.3    Foley, C.4    Mockridge, J.W.5    Wright, M.J.6
  • 22
    • 0036839039 scopus 로고    scopus 로고
    • Molecular pathophysiology of myofiber injury in deficiencies of the dystrophin-glycoprotein complex
    • B.J. Petrof Molecular pathophysiology of myofiber injury in deficiencies of the dystrophin-glycoprotein complex Am J Phys Med Rehabil 81 2002 S162 S174
    • (2002) Am J Phys Med Rehabil , vol.81
    • Petrof, B.J.1
  • 23
    • 0031939753 scopus 로고    scopus 로고
    • Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family
    • Y. Wang, S. Huang, V.P. Sah, J. Ross Jr., J.H. Brown, and J. Han Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family J Biol Chem 273 1998 2161 2168
    • (1998) J Biol Chem , vol.273 , pp. 2161-2168
    • Wang, Y.1    Huang, S.2    Sah, V.P.3    Ross Jr., J.4    Brown, J.H.5    Han, J.6
  • 26
    • 0030749330 scopus 로고    scopus 로고
    • A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression
    • D. Zechner, D.J. Thuerauf, D.S. Hanford, P.M. McDonough, and C.C. Glembotski A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression J Cell Biol 139 1997 115 127
    • (1997) J Cell Biol , vol.139 , pp. 115-127
    • Zechner, D.1    Thuerauf, D.J.2    Hanford, D.S.3    McDonough, P.M.4    Glembotski, C.C.5
  • 27
    • 85047687695 scopus 로고    scopus 로고
    • The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis
    • S. Zhang, C. Weinheimer, M. Courtois, A. Kovacs, C.E. Zhang, and A.M. Cheng The role of the Grb2-p38 MAPK signaling pathway in cardiac hypertrophy and fibrosis J Clin Invest 111 2003 833 841
    • (2003) J Clin Invest , vol.111 , pp. 833-841
    • Zhang, S.1    Weinheimer, C.2    Courtois, M.3    Kovacs, A.4    Zhang, C.E.5    Cheng, A.M.6
  • 28
    • 0033818186 scopus 로고    scopus 로고
    • Mutations in the human delta-sarcoglycan gene in familial and sporadic dilated cardiomyopathy
    • S. Tsubata, K.R. Bowles, M. Vatta, C. Zintz, J. Titus, and L. Muhonen Mutations in the human delta-sarcoglycan gene in familial and sporadic dilated cardiomyopathy J Clin Invest 106 2000 655 662
    • (2000) J Clin Invest , vol.106 , pp. 655-662
    • Tsubata, S.1    Bowles, K.R.2    Vatta, M.3    Zintz, C.4    Titus, J.5    Muhonen, L.6
  • 29
    • 0033017374 scopus 로고    scopus 로고
    • Enteroviral protease 2A cleaves dystrophin: Evidence of cytoskeletal disruption in an acquired cardiomyopathy
    • C. Badorff, G.H. Lee, B.J. Lamphear, M.E. Martone, K.P. Campbell, and R.E. Rhoads Enteroviral protease 2A cleaves dystrophin: evidence of cytoskeletal disruption in an acquired cardiomyopathy Nat Med 5 1999 320 326
    • (1999) Nat Med , vol.5 , pp. 320-326
    • Badorff, C.1    Lee, G.H.2    Lamphear, B.J.3    Martone, M.E.4    Campbell, K.P.5    Rhoads, R.E.6


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