The uncoupling agent 2,4-dinitrophenol improves mitochondrial homeostasis following striatal quinolinic acid injections

Journal of Neurotrauma

Volumn 22, Issue 10, 2005, Pages 1142-1149

  Korde, Amit S ^a   Sullivan, Patrick G ^a   Maragos, William F ^a,b,c  

^a UNIVERSITY OF KENTUCKY   (United States)

^b UNIVERSITY OF KENTUCKY   (United States)

^c UNIVERSITY OF KENTUCKY MEDICAL CENTER   (United States)

Author keywords

Acute injury; Calcium; Mitochondrial uncoupling; Oxymetry; Reactive oxygen species; Respiratory control ratio

Indexed keywords

2,4 DINITROPHENOL; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; QUINOLINIC ACID; UNCOUPLING PROTEIN;

ANIMAL EXPERIMENT; ARTICLE; BRAIN INJURY; CALCIUM TRANSPORT; CONTROLLED STUDY; ENERGY TRANSFER; EXCITOTOXICITY; HOMEOSTASIS; MALE; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL RESPIRATION; NERVE CELL NECROSIS; NEUROPROTECTION; NONHUMAN; RAT;

2,4-DINITROPHENOL; ANIMALS; CELL RESPIRATION; CORPUS STRIATUM; EXCITATORY AMINO ACID AGONISTS; HOMEOSTASIS; INJECTIONS, INTRAVENTRICULAR; MALE; MITOCHONDRIA; QUINOLINIC ACID; RATS; RATS, SPRAGUE-DAWLEY; REACTIVE OXYGEN SPECIES; UNCOUPLING AGENTS;

EID: 27444431612     PISSN: 08977151     EISSN: None     Source Type: Journal    
DOI: 10.1089/neu.2005.22.1142     Document Type: Article

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