Na+ overload-induced mitochondrial damage in the ischemic heart

Canadian Journal of Physiology and Pharmacology

Volumn 82, Issue 12, 2004, Pages 1033-1043

  Takeo, Satoshi ^a   Tanonaka, Kouichi ^a  

^a TOKYO UNIVERSITY OF PHARMACY AND LIFE SCIENCES   (Japan)

Author keywords

Mitochondria; Myocardial ischemia; Na + H+ exchanger; Na+ channels; Na+ overload

Indexed keywords

2 [2 [4 (4 NITROBENZYLOXY)PHENYL]ETHYL]ISOTHIOUREA METHANESULFONATE; 2 [4 (2,5 DIFLUOROBENZYLOXY)PHENOXY] 5 ETHOXYANILINE; 5 (N ETHYL N ISOPROPYL)AMILORIDE; AMILORIDE; ANTIARRHYTHMIC AGENT; BLOCKING AGENT; CARDIAC AGENT; CARIPORIDE; FREE RADICAL; GLIBENCLAMIDE; SODIUM CHANNEL; SODIUM CHANNEL BLOCKING AGENT; SODIUM ION; SODIUM PROTON EXCHANGE PROTEIN; SODIUM PROTON EXCHANGE PROTEIN 1; SODIUM PROTON EXCHANGE PROTEIN INHIBITOR; TETRODOTOXIN; UNCLASSIFIED DRUG;

CELL FUNCTION; CORRELATION ANALYSIS; DISORDERS OF MITOCHONDRIAL FUNCTIONS; HEART DISEASE; HEART FUNCTION; HEART MUSCLE CONTRACTILITY; HEART MUSCLE ISCHEMIA; HEART PROTECTION; HOMEOSTASIS; HUMAN; MITOCHONDRION; NONHUMAN; POTASSIUM DEPLETION; PRIORITY JOURNAL; REPERFUSION INJURY; REVIEW;

ANIMALS; ENERGY METABOLISM; MITOCHONDRIA, HEART; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; POTASSIUM; RATS; SODIUM; SODIUM CHANNEL BLOCKERS; SODIUM-CALCIUM EXCHANGER;

EID: 14844320726     PISSN: 00084212     EISSN: None     Source Type: Journal    
DOI: 10.1139/y04-124     Document Type: Review

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