Oncogenic K-RAS is required to maintain changes in cytoskeletal organization, adhesion, and motility in colon cancer cells

Cancer Research

Volumn 65, Issue 4, 2005, Pages 1244-1250

  Pollock, Claire B ^a,d   Shirasawa, Senji ^c   Sasazuki, Takehiko ^c   Kolch, Walter ^a,b   Dhillon, Amardeep S ^a  

^a BEATSON INSTITUTE FOR CANCER RESEARCH   (United Kingdom)

^b UNIVERSITY OF GLASGOW   (United Kingdom)

^c NATIONAL CENTER FOR GLOBAL HEALTH AND MEDICINE   (Japan)

^d NATIONAL CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

K RAS PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE; RHO FACTOR; TRANSCRIPTION FACTOR AP 1; TRANSCRIPTION FACTOR FRA 1;

ARTICLE; CANCER CELL; CELL ADHESION; CELL DISRUPTION; CELL MOTILITY; COLON CANCER; CONTROLLED STUDY; CYTOSKELETON; DOWNSTREAM PROCESSING; EXTRACELLULAR MATRIX; FOCAL ADHESION; GENE DELETION; GENE TARGETING; HUMAN; HUMAN CELL; METASTASIS; ONCOGENE K RAS; PRIORITY JOURNAL; PROTEIN FUNCTION; SIGNAL TRANSDUCTION; STRESS FIBER;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ACTINS; CELL ADHESION; CELL MOVEMENT; CELL TRANSFORMATION, NEOPLASTIC; COLONIC NEOPLASMS; CYTOSKELETON; GENE DELETION; GENES, RAS; HCT116 CELLS; HUMANS; MAP KINASE KINASE KINASES; MAP KINASE SIGNALING SYSTEM; PROTO-ONCOGENE PROTEINS C-FOS; TRANSFECTION;

EID: 13944249516     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: 10.1158/0008-5472.CAN-04-1911     Document Type: Article

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