Long-Term caspase inhibition ameliorates apoptosis, reduces myocardial troponin-I cleavage, protects left ventricular function, and attenuates remodeling in rats with myocardial infarction

Journal of the American College of Cardiology

Volumn 43, Issue 2, 2004, Pages 295-301

  Chandrashekhar, Y ^a,b   Sen, Soma ^a   Anway, Ruth ^a   Shuros, Allan ^a   Anand, Inder ^a  

^a UNIVERSITY OF MINNESOTA   (United States)

^b UNIVERSITY OF MINNESOTA MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE; CASPASE 3; CASPASE INHIBITOR; PEPTIDE CHLOROMETHYL KETONE; TROPONIN I; UNCLASSIFIED DRUG; Z ASP 2,6 DICHLOROBENZOYL OXYMETHYLKETONE; Z-ASP-2,6-DICHLOROBENZOYL-OXYMETHYLKETONE;

ANIMAL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CARDIOPULMONARY HEMODYNAMICS; CONGESTIVE HEART FAILURE; CONTROLLED STUDY; DRUG EFFECT; ENZYME ACTIVATION; ENZYME INHIBITION; EVALUATION; HEART AUTOMATICITY; HEART INFARCTION; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE CELL; HEART MUSCLE NECROSIS; HEART PROTECTION; HEART VENTRICLE REMODELING; HEART WEIGHT; HEMODYNAMICS; MALE; MORPHOMETRICS; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; RAT; RAT STRAIN; SHAM FEEDING; SPECIES COMPARISON;

EID: 1042299088     PISSN: 07351097     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.jacc.2003.09.026     Document Type: Article

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