Cross-Priming of Diabetogenic T Cells Dissociated from CTL-Induced Shedding of β Cell Autoantigens

Journal of Immunology

Volumn 171, Issue 12, 2003, Pages 6900-6909

  Yamanouchi, Jun ^a   Verdaguer, Joan ^a,b   Han, Bingye ^a   Amrani, Abdelaziz ^a,c   Serra, Pau ^a   Santamaria, Pere ^a,d  

^a UNIVERSITY OF CALGARY   (Canada)

^b HOSPITAL UNIVERSITARI GERMANS TRIAS I PUJOL   (Spain)

^c UNIVERSITY OF SHERBROOKE   (Canada)

^d UNIVERSITY OF CALGARY   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIGEN; CD7 ANTIGEN; FAS ANTIGEN; FASL PROTEIN; LIGAND; LYMPHOCYTE ANTIGEN RECEPTOR; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; MEMBRANE PROTEIN; T LYMPHOCYTE RECEPTOR; UNCLASSIFIED DRUG;

ADENOVIRUS; ANIMAL; ANIMAL CELL; ANTIGEN PRESENTATION; ANTIGEN PRESENTING CELL; ARTICLE; AUTOIMMUNITY; B LYMPHOCYTE; BIOSYNTHESIS; CELL COMMUNICATION; CELL INTERACTION; CELL MOTION; CONTROLLED STUDY; CROSS BREEDING; CYTOLYSIS; CYTOTOXIC T LYMPHOCYTE; CYTOTOXICITY; DIABETES MELLITUS; DIABETOGENESIS; DISEASE COURSE; FEMALE; GENE EXPRESSION; GENETICS; IMMUNOLOGY; INFLAMMATION; INSULIN DEPENDENT DIABETES MELLITUS; INTERPHASE; LYMPH NODE; LYMPHOCYTE ACTIVATION; MALE; METABOLISM; MOUSE; MOUSE STRAIN; NONHUMAN; PANCREAS ISLET; PATHOLOGY; PRIORITY JOURNAL; PROTEIN TRANSPORT; T LYMPHOCYTE; TRANSGENE; TRANSGENIC MOUSE;

EID: 0346366975     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.171.12.6900     Document Type: Article

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