Epithelial Na+ channel mutants causing Liddle's syndrome retain ability to respond to aldosterone and vasopressin

American Journal of Physiology - Renal Physiology

Volumn 285, Issue 3 54-3, 2003, Pages

  Auberson, Muriel ^a   Hoffmann Pochon, Nicole ^a   Vandewalle, A ^b   Kellenberger, Stephan ^a   Schild, Laurent ^a,c  

^a UNIVERSITY OF LAUSANNE   (Switzerland)

^b INSERM   (France)

^c University of Lausanne Departement de Pharmacologie et de Toxicologie   (Switzerland)

Author keywords

Collecting duct; Hypertension; PY motif

Indexed keywords

ALDOSTERONE; PROTEIN SUBUNIT; RETROVIRUS VECTOR; SODIUM CHANNEL; VASOPRESSIN;

ANIMAL CELL; ARTICLE; CARBOXY TERMINAL SEQUENCE; CELL SURFACE; CONTROLLED STUDY; GENE MUTATION; GENE TRANSFER; HORMONAL REGULATION; HORMONE SENSITIVITY; HYPERTENSION; KIDNEY COLLECTING TUBULE; LIDDLE SYNDROME; MOUSE; MUTATIONAL ANALYSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN MOTIF; PROTEIN STABILITY; SHORT CIRCUIT CURRENT; SODIUM ABSORPTION; SODIUM TRANSPORT; WILD TYPE;

EID: 0041464464     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.00071.2003     Document Type: Article

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