Rescue of the skeletal phenotype in CasR-deficient mice by transfer onto the Gcm2 null background

Journal of Clinical Investigation

Volumn 111, Issue 7, 2003, Pages 1029-1037

  Tu, Qisheng ^a   Pi, Min ^a   Karsenty, Gerard ^b   Simpson, Leigh ^a   Liu, Shiguang ^a   Quarles, L Darryl ^a  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

^b BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM SENSING RECEPTOR; PARATHYROID HORMONE;

ANIMAL TISSUE; ARTICLE; BONE DEVELOPMENT; BONE MINERALIZATION; CARTILAGE; CHONDROGENESIS; CONTROLLED STUDY; GENOTYPE; GLIA CELL; HOMOZYGOSITY; HYPERTHYROIDISM; LETHALITY; NONHUMAN; NULL ALLELE; OSTEOMALACIA; PARATHYROID GLAND; PHENOTYPE; PRIORITY JOURNAL; RICKETS; SKELETON;

ANIMALS; BONE AND BONES; CALCIUM; DENSITOMETRY; EXONS; GENOTYPE; HETEROZYGOTE; HOMOZYGOTE; HYPERPARATHYROIDISM; KIDNEY; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MICROSCOPY, FLUORESCENCE; NEUROPEPTIDES; OSTEOMALACIA; PHENOTYPE; RECEPTORS, CALCIUM-SENSING; RECEPTORS, CELL SURFACE; RICKETS; TIME FACTORS; TRANS-ACTIVATORS;

EID: 0037382146     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI200317054     Document Type: Article

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