Autoimmune islet destruction in spontaneous type 1 diabetes is not β-cell exclusive

Nature Medicine

Volumn 9, Issue 2, 2003, Pages 198-205

  Winer, Shawn ^a   Tsui, Hubert ^a   Lau, Ambrose ^a   Song, Aihua ^a   Li, Xiaomao ^b   Cheung, Roy K ^a   Sampson, Anastazia ^a   Afifiyan, Fatemeh ^a   Elford, Alisha ^c   Jackowski, George ^b   Becker, Dorothy J ^d   Santamaria, Pere ^e   Ohashi, Pamela ^c   Dosch, H Michael ^a  

^a HOSPITAL FOR SICK CHILDREN   (Canada)

^b Syn X Pharma Inc   (Canada)

^c PRINCESS MARGARET HOSPITAL   (Canada)

^d CHILDREN S HOSPITAL OF PITTSBURGH   (United States)

^e UNIVERSITY OF CALGARY   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; CD8 ANTIGEN; GLIAL FIBRILLARY ACIDIC PROTEIN; INSULIN; PANCREAS ISLET CELL ANTIBODY; PROTEIN S100B; T LYMPHOCYTE RECEPTOR;

ADOLESCENT; ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOIMMUNITY; B LYMPHOCYTE; BYSTANDER EFFECT; CELL DEATH; CELL DESTRUCTION; FEMALE; GENE TRANSFER; IMMUNE RESPONSE; IMMUNOTHERAPY; IMPAIRED GLUCOSE TOLERANCE; INFLAMMATION; INSULIN DEPENDENT DIABETES MELLITUS; INSULITIS; MALE; MOUSE; MOUSE STRAIN; NONHUMAN; PANCREAS ISLET BETA CELL; PANCREAS ISLET CELL; PRIORITY JOURNAL; SCHWANN CELL; SCID MOUSE; T LYMPHOCYTE; TRANSGENE; TRANSGENIC MOUSE;

ANIMALS; AUTOANTIBODIES; BASE SEQUENCE; DIABETES MELLITUS, TYPE 1; DNA PRIMERS; FEMALE; FLUORESCENT ANTIBODY TECHNIQUE; GLIAL FIBRILLARY ACIDIC PROTEIN; ISLETS OF LANGERHANS; MALE; MICE; MICE, INBRED STRAINS; NERVE GROWTH FACTORS; S100 PROTEINS; SCHWANN CELLS; SPECIES SPECIFICITY;

ANIMALIA; LYMPHOCYTIC CHORIOMENINGITIS VIRUS; MUS MUSCULUS;

EID: 0037327274     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/nm818     Document Type: Article

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