Disruption of gap junctions in toxicity and carcinogenicity

Toxicological Sciences

Volumn 71, Issue 2, 2003, Pages 146-153

  Chipman, J Kevin ^a   Mally, Angela ^a   Edwards, Gareth O ^a  

^a UNIVERSITY OF BIRMINGHAM   (United Kingdom)

Author keywords

Bystander; Carcinogenesis; Connexin; Gap junction; Intercellular communication

Indexed keywords

1,1 DICHLORO 2,2 BIS(4 CHLOROPHENYL)ETHYLENE; 2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; ANESTHETIC AGENT; ANTISENSE OLIGONUCLEOTIDE; CALCIUM ION; CARCINOGEN; CHLORPHENOTANE; CLOFIBRATE; CONNEXIN 43; DOXORUBICIN; ETOPOSIDE; GANCICLOVIR; GAP JUNCTION PROTEIN; HALOTHANE; HEPTACHLOR; HEPTANOL; LINDANE; METHAPYRILENE; OCTANOL; PACLITAXEL; PEROXISOME PROLIFERATOR; PESTICIDE; PHENOBARBITAL; PHORBOL ESTER; PIRINIXIC ACID; PROPYLTHIOURACIL; SUPEROXIDE; THIOACETAMIDE; THYMIDINE KINASE; UNINDEXED DRUG;

APOPTOSIS; BYSTANDER EFFECT; CANCER INHIBITION; CANCER PREVENTION; CARCINOGENICITY; CELL COMMUNICATION; CELL PROLIFERATION; CHEMICAL CARCINOGENESIS; CHEMICAL INJURY; CHEMOPROPHYLAXIS; GAP JUNCTION; GENE THERAPY; HOMEOSTASIS; HUMAN; METASTASIS; NONHUMAN; PROTEIN FUNCTION; PROTEIN TARGETING; RADIATION INJURY; REVIEW;

ANIMALS; CARCINOGENS; CELLS, CULTURED; CONNEXINS; DOWN-REGULATION; GAP JUNCTIONS; HUMANS;

EID: 0037292354     PISSN: 10966080     EISSN: None     Source Type: Journal    
DOI: 10.1093/toxsci/71.2.146     Document Type: Review

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