SCOPUS 정보 검색 플랫폼

Volumn 515, Issue 1-3, 2002, Pages 119-126

Differential activation of nuclear factor-κB by tumour necrosis factor receptor subtypes. TNFR1 predominates whereas TNFR2 activates transcription poorly

(7) McFarlane, Shona M a Pashmi, Ghazaleh a Connell, Michelle C a Littlejohn, Alison F a Tucker, Steven J a Vandenabeele, Peter b MacEwan, David J a

a UNIVERSITY OF ABERDEEN (United Kingdom)

b DEPARTMENT OF PLANT SYSTEMS BIOLOGY (Belgium)

Author keywords

Cytokine; Kinase; Receptor; Signal transduction; Subtype; Tumour

Indexed keywords

I KAPPA B ALPHA; I KAPPA B BETA; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; RECEPTOR SUBTYPE; TUMOR NECROSIS FACTOR RECEPTOR; TUMOR NECROSIS FACTOR RECEPTOR 1; TUMOR NECROSIS FACTOR RECEPTOR 2;

ARTICLE; BIOASSAY; CONTROLLED STUDY; CYTOTOXICITY; ELECTROPHORETIC MOBILITY; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN EXPRESSION; REGULATORY MECHANISM; REPORTER GENE; SIGNAL TRANSDUCTION;

ANTIGENS, CD; CELL LINE; DNA-BINDING PROTEINS; ELECTROPHORETIC MOBILITY SHIFT ASSAY; GENES, REPORTER; HELA CELLS; HUMANS; I-KAPPA B PROTEINS; IMMUNE SERA; JNK MITOGEN-ACTIVATED PROTEIN KINASES; KIDNEY; MITOGEN-ACTIVATED PROTEIN KINASES; NF-KAPPA B; RECEPTORS, TUMOR NECROSIS FACTOR; RECEPTORS, TUMOR NECROSIS FACTOR, TYPE I; RECEPTORS, TUMOR NECROSIS FACTOR, TYPE II; RECOMBINANT FUSION PROTEINS; SIGNAL TRANSDUCTION; TRANSCRIPTION, GENETIC; TRANSFECTION;

EID: 0037181492 PISSN: 00145793 EISSN: None Source Type: Journal
DOI: 10.1016/S0014-5793(02)02450-X Document Type: Article

Times cited : (80)

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