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Oncogene

Volumn 21, Issue 32, 2002, Pages 5002-5005

Impaired DNA damage-induced nuclear Rad51 foci formation uniquely characterizes Fanconi anemia group D1

(4) Godthelp, Barbara C a Artwert, Fré b Joenje, Hans b Zdzienicka, Małgorzata Z a,c

a LEIDEN UNIVERSITY MEDICAL CENTER (Netherlands)

b VU UNIVERSITY MEDICAL CENTER (Netherlands)

c NICOLAUS COPERNICUS UNIVERSITY (Poland)

Author keywords

Fanconi anemia; Homologous recombination; MMC; Nuclear accumulation; Rad51 foci

Indexed keywords

DOUBLE STRANDED DNA; FANCONI ANEMIA PROTEIN; GENE PRODUCT; MITOMYCIN C; PROTEIN; RAD51 PROTEIN; UNCLASSIFIED DRUG;

ARTICLE; CANCER SUSCEPTIBILITY; CHROMOSOME DISORDER; CONTROLLED STUDY; DNA CROSS LINKING; DNA DAMAGE; DNA REPAIR; FANCONI ANEMIA; GENETIC RECOMBINATION; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; PHENOTYPE; PRIORITY JOURNAL; PROTEIN ASSEMBLY; PROTEIN STABILITY; RADIATION EXPOSURE; X IRRADIATION;

CELL NUCLEUS; CELLS, CULTURED; DNA DAMAGE; DNA-BINDING PROTEINS; FANCONI ANEMIA; FIBROBLASTS; HUMANS; MITOMYCIN; NUCLEIC ACID SYNTHESIS INHIBITORS; RAD51 RECOMBINASE; RECOMBINATION, GENETIC;

EID: 0037173719 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205656 Document Type: Article

Times cited : (92)

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