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Volumn 277, Issue 21, 2002, Pages 18640-18648

α-thrombin induces rapid and sustained Akt phosphorylation by β-arrestin1-dependent and -independent mechanisms, and only the sustained Akt phosphorylation is essential for G1 phase progression

(4) Goel, Reema a Phillips Mason, Polly J a Raben, Daniel M a Baldassare, Joseph J a,b

a SAINT LOUIS UNIVERSITY SCHOOL OF MEDICINE (United States)

b JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL CULTURE; ENZYME INHIBITION; ENZYMES; MUTAGENESIS;

CELL CYCLE;

BIOCHEMISTRY;

BETA ARRESTIN; CYCLIN D1; CYCLIN DEPENDENT KINASE 4; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN KINASE B; THROMBIN;

ANIMAL CELL; ARTICLE; CELL CYCLE G1 PHASE; CHINESE HAMSTER; CONTROLLED STUDY; DNA SYNTHESIS; ENZYME ACTIVITY; FIBROBLAST; NONHUMAN; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; ARRESTINS; CHO CELLS; CRICETINAE; ENZYME ACTIVATION; G1 PHASE; MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; THROMBIN;

ANIMALIA; CRICETINAE; CRICETULUS GRISEUS;

EID: 0037166254 PISSN: 00219258 EISSN: None Source Type: Journal
DOI: 10.1074/jbc.M108995200 Document Type: Article

Times cited : (85)

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