Radiation induction of p53 in cells from Nijmegen breakage syndrome is defective but not similar to ataxia-telangiectasia

Biochemical and Biophysical Research Communications

Volumn 242, Issue 3, 1998, Pages 602-607

  Matsuura, Kanji ^a,b   Balmukhanov, Timur ^c   Tauchi, Hiroshi ^a   Weemaes, Corry ^d   Smeets, Domique ^d   Chrzanowska, Krystyna ^e   Endou, Satoru ^a   Matsuura, Shinnya ^a   Komatsu, Kenshi ^a  

^a HIROSHIMA UNIVERSITY   (Japan)

^b HIROSHIMA UNIV SCHOOL OF MEDICINE   (Japan)

^c Inst of Molec Biol and B   (Kazakhstan)

^d RADBOUD UNIVERSITY MEDICAL CENTER   (Netherlands)

^e CHILDREN'S MEMORIAL HEALTH INSTITUTE   (Poland)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN P21; PROTEIN P53;

ARTICLE; ATAXIA TELANGIECTASIA; CELL CYCLE; CONTROLLED STUDY; DNA DAMAGE; FIBROBLAST; HUMAN; HUMAN CELL; IONIZING RADIATION; NIJMEGEN BREAKAGE SYNDROME; PRIORITY JOURNAL; PROTEIN INDUCTION; RADIOSENSITIVITY; SIGNAL TRANSDUCTION; TIME; TUMOR SUPPRESSOR GENE;

ATAXIA; ATAXIA TELANGIECTASIA;

EID: 0032567642     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.1997.7924     Document Type: Article

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