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Volumn 21, Issue 30, 2002, Pages 4680-4690
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The cyclin D1 high and cyclin E high subgroups of breast cancer: Separate pathways in tumorogenesis based on pattern of genetic aberrations and inactivation of the pRb node
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Author keywords
Breast cancer; cdk inhibitors; Cell cycle; Cyclins; pRb pathway; Proliferation
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Indexed keywords
CYCLIN D1;
CYCLIN DEPENDENT KINASE INHIBITOR;
CYCLIN E;
MYC PROTEIN;
PROTEIN BCL 2;
PROTEIN P21;
PROTEIN P27;
PROTEIN P53;
REGULATOR PROTEIN;
RETINOBLASTOMA PROTEIN;
ADULT;
AGED;
ARTICLE;
BREAST CARCINOGENESIS;
BREAST CARCINOMA;
CANCER MODEL;
CANCER TISSUE;
CELL CYCLE;
CELL PROLIFERATION;
CONTROLLED STUDY;
ENZYME ACTIVITY;
ENZYME ASSAY;
FEMALE;
GENE AMPLIFICATION;
GENE MUTATION;
GENE OVEREXPRESSION;
GENETIC TRANSFECTION;
HUMAN;
HUMAN CELL;
HUMAN TISSUE;
MAJOR CLINICAL STUDY;
MALIGNANT TRANSFORMATION;
ONCOGENE C ERB;
PRIORITY JOURNAL;
PROTEIN ANALYSIS;
PROTEIN DEFECT;
PROTEIN DETERMINATION;
PROTEIN FUNCTION;
PROTEIN INDUCTION;
PROTEIN PHOSPHORYLATION;
REGULATORY MECHANISM;
TISSUE LEVEL;
BLOTTING, WESTERN;
BREAST NEOPLASMS;
CELL DIVISION;
CYCLIN D1;
CYCLIN E;
CYCLIN-DEPENDENT KINASES;
FOLLOW-UP STUDIES;
HUMANS;
IN SITU HYBRIDIZATION, FLUORESCENCE;
MODELS, BIOLOGICAL;
RECEPTORS, ESTROGEN;
RETINOBLASTOMA PROTEIN;
SIGNAL TRANSDUCTION;
SURVIVAL RATE;
TUMOR CELLS, CULTURED;
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EID: 0037063165
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1205578 Document Type: Article |
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Times cited : (96)
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References (48)
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