The promoter context determines mutual repression or synergism between NF-κB and the glucocorticoid receptor

Biological Chemistry

Volumn 383, Issue 12, 2002, Pages 1947-1951

  Hofmann, Thomas G ^a   Lienhard Schmitz, M ^b  

^a LEIBNIZ INSTITUTE FOR EXPERIMENTAL VIROLOGY   (Germany)

^b UNIVERSITY OF BERN   (Switzerland)

Author keywords

Crosstalk; Gene expression; Glucocorticoid receptor; Inflammation; NF B

Indexed keywords

DEXAMETHASONE; GLUCOCORTICOID RECEPTOR; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; TUMOR NECROSIS FACTOR ALPHA;

ARTICLE; BINDING SITE; CONTROLLED STUDY; DNA RESPONSIVE ELEMENT; GENE CONSTRUCT; GENE REPRESSION; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN BINDING; PROTEIN FUNCTION; TRANSCRIPTION INITIATION;

BINDING SITES; DNA; HUMANS; LUCIFERASES; NF-KAPPA B; PROMOTER REGIONS (GENETICS); RECEPTORS, GLUCOCORTICOID; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0036925189     PISSN: 14316730     EISSN: None     Source Type: Journal    
DOI: 10.1515/BC.2002.219     Document Type: Article

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