CTLA-4 and autoimmune thyroid disease: Lack of influence of the A49G signal peptide polymorphism on functional recombinant human CTLA-4

Cellular Immunology

Volumn 215, Issue 2, 2002, Pages 133-140

  Xu, Yang ^a   Graves, Peter N ^a   Tomer, Yaron ^a   Davies, Terry F ^a  

^a MOUNT SINAI SCHOOL OF MEDICINE   (United States)

Author keywords

CTLA 4; IL 2; Jurkat T cells; SNP; Thyroid

Indexed keywords

ANTIBODY; CYTOTOXIC T LYMPHOCYTE ANTIGEN 4; INTERLEUKIN 2; PHYTOHEMAGGLUTININ; RECOMBINANT ANTIGEN; SIGNAL PEPTIDE;

ALLELE; ANTIGEN ANTIBODY COMPLEX; ANTIGEN EXPRESSION; ANTIGEN FUNCTION; ARTICLE; AUTOIMMUNE DISEASE; CELL MEMBRANE; CONCENTRATION RESPONSE; CONTROLLED STUDY; CYTOKINE PRODUCTION; FLUORESCENCE ACTIVATED CELL SORTING; GENE LINKAGE DISEQUILIBRIUM; GENETIC CODE; GENETIC LINKAGE; GENETIC TRANSCRIPTION; GENETIC TRANSFECTION; HUMAN; HUMAN CELL; LEUKEMIA CELL LINE; MOLECULAR CLONING; MOLECULAR MECHANICS; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN DETERMINATION; QUANTITATIVE ANALYSIS; SINGLE NUCLEOTIDE POLYMORPHISM; THYROID DISEASE;

ALLELES; ANTIBODIES, MONOCLONAL; ANTIGENS; ANTIGENS, CD; ANTIGENS, DIFFERENTIATION; CELL SEPARATION; FLOW CYTOMETRY; GENES, REPORTER; HUMANS; IMMUNOCONJUGATES; IMMUNOGLOBULIN FC FRAGMENTS; IMMUNOSUPPRESSIVE AGENTS; INTERLEUKIN-2; JURKAT CELLS; POLYMORPHISM, SINGLE NUCLEOTIDE; PROTEIN SORTING SIGNALS; RECOMBINANT FUSION PROTEINS; THYROIDITIS, AUTOIMMUNE;

EID: 0036389259     PISSN: 00088749     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0008-8749(02)00018-7     Document Type: Article

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