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Volumn 66, Issue 4, 2002, Pages 552-560
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Effect of lack of Interleukin-4, Interleukin-12, Interleukin-18, or the Interferon-γ receptor on virus replication, cytokine response, and lung pathology during respiratory syncytial virus infection in mice
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Author keywords
IFN ; Lung pathology; Mouse; RSV; Th2 response
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Indexed keywords
CYTOKINE;
GAMMA INTERFERON;
GAMMA INTERFERON RECEPTOR;
INTERLEUKIN 12;
INTERLEUKIN 13;
INTERLEUKIN 18;
INTERLEUKIN 4;
INTERLEUKIN 5;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BRONCHIOLITIS;
CELL INFILTRATION;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
DISEASE ASSOCIATION;
EOSINOPHIL;
FEMALE;
HISTOPATHOLOGY;
IMMUNOPATHOGENESIS;
LUNG ALVEOLITIS;
MOUSE;
NONHUMAN;
PROTEIN EXPRESSION;
RESPIRATORY SYNCYTIAL PNEUMOVIRUS;
SIGNAL TRANSDUCTION;
TH1 CELL;
TH2 CELL;
VIRUS INFECTION;
VIRUS LOAD;
VIRUS PATHOGENESIS;
VIRUS REPLICATION;
ANIMALS;
CYTOKINES;
FEMALE;
HUMANS;
INTERLEUKIN-12;
INTERLEUKIN-18;
INTERLEUKIN-4;
LUNG;
MICE;
MICE, KNOCKOUT;
RECEPTORS, INTERFERON;
RESPIRATORY SYNCYTIAL VIRUS INFECTIONS;
RESPIRATORY SYNCYTIAL VIRUSES;
TH1 CELLS;
TH2 CELLS;
VIRUS REPLICATION;
RESPIRATORY SYNCYTIAL VIRUS;
RICE STRIPE VIRUS;
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EID: 0036200715
PISSN: 01466615
EISSN: None
Source Type: Journal
DOI: 10.1002/jmv.2180 Document Type: Article |
Times cited : (42)
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References (25)
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