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Volumn 66, Issue 4, 2002, Pages 552-560

Effect of lack of Interleukin-4, Interleukin-12, Interleukin-18, or the Interferon-γ receptor on virus replication, cytokine response, and lung pathology during respiratory syncytial virus infection in mice

Author keywords

IFN ; Lung pathology; Mouse; RSV; Th2 response

Indexed keywords

CYTOKINE; GAMMA INTERFERON; GAMMA INTERFERON RECEPTOR; INTERLEUKIN 12; INTERLEUKIN 13; INTERLEUKIN 18; INTERLEUKIN 4; INTERLEUKIN 5;

EID: 0036200715     PISSN: 01466615     EISSN: None     Source Type: Journal    
DOI: 10.1002/jmv.2180     Document Type: Article
Times cited : (42)

References (25)
  • 25
    • 0034163312 scopus 로고    scopus 로고
    • IL-12 independent Th1-type immune responses to respiratory viral infection: Requirement of IL-18 for IFNγ release in the lung but not for the differentiation of viral-reactive Th1-type lymphocytes
    • (2000) J Immunol , vol.164 , pp. 2575-2584
    • Xing, Z.1    Zganiacz, A.2    Wang, J.3    Divangahi, M.4    Nawaz, F.5


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.