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Volumn 22, Issue 1, 2002, Pages 59-71
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Diabetes teratogenicity in mice is accompanied with distorted expression of TGF-β2 in the uterus
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Author keywords
[No Author keywords available]
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Indexed keywords
GAMMA INTERFERON;
INTERLEUKIN 1;
MESSENGER RNA;
TRANSFORMING GROWTH FACTOR BETA;
TRANSFORMING GROWTH FACTOR BETA2;
TUMOR NECROSIS FACTOR ALPHA;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CONGENITAL MALFORMATION;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
DOSE TIME EFFECT RELATION;
EMBRYO DEATH;
EMBRYOTOXICITY;
FEMALE;
FETUS MALFORMATION;
IMMUNOHISTOCHEMISTRY;
IMMUNOPOTENTIATION;
IMMUNOTHERAPY;
IN SITU HYBRIDIZATION;
MOUSE;
NONHUMAN;
PREGNANCY DIABETES MELLITUS;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RAT;
REPRODUCTIVE TOXICITY;
RNA ANALYSIS;
STREPTOZOCIN DIABETES;
SURVIVAL;
TERATOGENICITY;
UTERUS;
ANIMALS;
DIABETES MELLITUS, EXPERIMENTAL;
EMBRYO LOSS;
FEMALE;
GENE EXPRESSION REGULATION;
IMMUNOENZYME TECHNIQUES;
IN SITU HYBRIDIZATION;
MICE;
MICE, INBRED ICR;
PREGNANCY;
RATS;
RATS, LONG-EVANS;
RNA, MESSENGER;
TIME FACTORS;
TRANSFORMING GROWTH FACTOR BETA;
TRANSFORMING GROWTH FACTOR BETA2;
UTERUS;
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EID: 0036135246
PISSN: 02703211
EISSN: None
Source Type: Journal
DOI: 10.1002/tcm.1039 Document Type: Article |
Times cited : (19)
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References (32)
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