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Volumn 3, Issue 1, 1997, Pages 27-33
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In vivo evidence for the existence of a threshold for hyperglycemia-induced major fetal malformations: relevance to the etiology of diabetic teratogenesis.
a a a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
GLYCOSYLATED HEMOGLOBIN;
STREPTOZOCIN;
ANIMAL;
ARTICLE;
BLOOD;
BREEDING;
CONGENITAL MALFORMATION;
EXPERIMENTAL DIABETES MELLITUS;
FEMALE;
GLUCOSE BLOOD LEVEL;
HYPERGLYCEMIA;
INCIDENCE;
INSTITUTE FOR CANCER RESEARCH MOUSE;
MOUSE;
PATHOPHYSIOLOGY;
PHYSIOLOGY;
PREGNANCY;
PREGNANCY DIABETES MELLITUS;
PREGNANCY OUTCOME;
PREGNANCY RATE;
PRENATAL DEVELOPMENT;
ABNORMALITIES;
ANIMALS;
BLOOD GLUCOSE;
DIABETES MELLITUS, EXPERIMENTAL;
EMBRYONIC AND FETAL DEVELOPMENT;
FEMALE;
HEMOGLOBIN A, GLYCOSYLATED;
HYPERGLYCEMIA;
INCIDENCE;
MICE;
MICE, INBRED ICR;
PREGNANCY;
PREGNANCY IN DIABETICS;
PREGNANCY OUTCOME;
PREGNANCY RATE;
PREGNANCY, ANIMAL;
STREPTOZOCIN;
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EID: 0031087744
PISSN: 13544195
EISSN: None
Source Type: Journal
DOI: None Document Type: Article |
Times cited : (19)
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References (0)
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