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Volumn 269, Issue 5, 2002, Pages 1511-1517

Regulation of RAS in human platelets: Evidence that activation of RAS is not sufficient to lead to ERK1-2 phosphorylation

Author keywords

ERK; Glycoprotein VI; Platelet; Protein kinase C RAS; Signalling; Thrombin; Thrombopoietin

Indexed keywords

2 [1 (3 AMIDINOTHIOPROPYL) 1H INDOL 3 YL] 3 (1 METHYL 1H INDOL 3 YL)MALEIMIDE; CD36 ANTIGEN; CONVULXIN; CYTOKINE RECEPTOR; GUANINE NUCLEOTIDE BINDING PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE 1; PROTEIN; PROTEIN KINASE C INHIBITOR; RECEPTOR; THROMBIN; THROMBOPOIETIN; TYROSINE;

EID: 0036123693     PISSN: 00142956     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1432-1033.2002.02798.x     Document Type: Article
Times cited : (26)

References (31)
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    • The heterotrimeric G q protein-coupled angiotensin II receptor activates p21 ras via the tyrosine kinase-Shc-Grb2-Sos pathway in cardiac myocytes
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    • Sadoshima, J.1    Izumo, S.2
  • 20
    • 0031005028 scopus 로고    scopus 로고
    • Evidence for MEK-independent pathways regulating the prolonged activation of the ERK-MAP kinases
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    • Grammer, T.C.1    Blenis, J.2
  • 21


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.