Gene-dosage-sensitive genetic interactions between inversus viscerum (iv), nodal, and activin type IIB receptor (ActRIIB) genes in asymmetrical patterning of the visceral organs along the left-right axis

Developmental Dynamics

Volumn 224, Issue 3, 2002, Pages 279-290

  Oh, S Paul ^a,c   Li, En ^b  

^a UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

^b HARVARD MEDICAL SCHOOL   (United States)

^c UNIVERSITY OF FLORIDA   (United States)

Author keywords

Activin receptor; Heterotaxy; Inversus viscerum; Isomerism; Laterality; Nodal

Indexed keywords

ACTIVIN RECEPTOR; RECEPTOR SUBTYPE; TRANSFORMING GROWTH FACTOR BETA; ACTIVIN RECEPTOR 2; ACTIVIN RECEPTOR TYPE II B; ACTIVIN RECEPTOR TYPE II-B; PROTEIN NODAL;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CHIMERA; CONGENITAL MALFORMATION; CONTROLLED STUDY; DISEASE SEVERITY; EMBRYO; EMBRYO PATTERN FORMATION; FEMALE; GENE; GENE DOSAGE; GENE FUNCTION; GENE INTERACTION; GENE MUTATION; HEART RIGHT ATRIUM; HOMOZYGOTE; HYPOPLASIA; INFERIOR CAVA VEIN; INVERSUS VISCERUM GENE; MALE; MOUSE; NONHUMAN; PENETRANCE; PHENOTYPE; PRIORITY JOURNAL; PROTEIN FAMILY; SIGNAL TRANSDUCTION; SITUS INVERSUS; SPLEEN DISEASE; VISCERA; ANIMAL; BIOLOGICAL MODEL; GENE EXPRESSION REGULATION; GENETICS; HEMISPHERIC DOMINANCE; ISOMERISM; METABOLISM; MOUSE MUTANT; MUTATION; NEWBORN; TIME;

ACTIVIN RECEPTORS, TYPE II; ANIMAL; ANIMALS, NEWBORN; GENE DOSAGE; GENE EXPRESSION REGULATION, DEVELOPMENTAL; HOMOZYGOTE; ISOMERISM; LATERALITY; MICE; MICE, KNOCKOUT; MODELS, GENETIC; MUTATION; PHENOTYPE; SIGNAL TRANSDUCTION; SITUS INVERSUS; SUPPORT, U.S. GOV'T, P.H.S.; TIME FACTORS; TRANSFORMING GROWTH FACTOR BETA; ANIMALS; FUNCTIONAL LATERALITY;

EID: 0035986557     PISSN: 10588388     EISSN: None     Source Type: Journal    
DOI: 10.1002/dvdy.10103     Document Type: Article

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