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Volumn 20, Issue 3, 2001, Pages 320-328

Knock-in mice with a chimeric human/murine p53 gene develop normally and show wild-type p53 responses to DNA damaging agents: A new biomedical research tool

(6) Luo, Jun Li a Yang, Qin a Tong, Wei Min b Hergenhahn, Manfred a Wang, Zhao Qi b Hollstein, Monica a

a GERMAN CANCER RESEARCH CENTER DKFZ (Germany)

b INTERNATIONAL AGENCY FOR RESEARCH ON CANCER (France)

Author keywords

Gene targeting; Mutagenesis; p53 mouse model

Indexed keywords

COMPLEMENTARY DNA; PROTEIN P53;

ALLELE; ANIMAL CELL; APOPTOSIS; ARTICLE; CHIMERA; CONTROLLED STUDY; DNA DAMAGE; EMBRYO; EXON; GENE INDUCTION; GENE MUTATION; GENE SEQUENCE; GENE TARGETING; KNOCKOUT MOUSE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DNA BINDING;

ANIMALS; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CELLS, CULTURED; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DEXAMETHASONE; DNA DAMAGE; EMBRYONIC AND FETAL DEVELOPMENT; EXONS; GAMMA RAYS; GENE EXPRESSION REGULATION; HOMOZYGOTE; HUMANS; INTEGRASES; MICE; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; SPLEEN; THYMUS GLAND; TUMOR SUPPRESSOR PROTEIN P53; ULTRAVIOLET RAYS; VIRAL PROTEINS;

ANIMALIA; MURINAE;

EID: 0035905749 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204080 Document Type: Article

Times cited : (120)

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