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Oncogene

Volumn 20, Issue 27, 2001, Pages 3580-3584

Msh-2 suppresses in vivo mutation in a gene dose and lesion dependent manner

(4) Sansom, Owen J a,b Toft, Neil J a Winton, Douglas J c Clarke, Alan R a

a CARDIFF UNIVERSITY (United Kingdom)

b UNIVERSITY OF EDINBURGH (United Kingdom)

c UNIVERSITY OF CAMBRIDGE (United Kingdom)

Author keywords

Alkylation; Apoptosis; Cisplatin; Heterozygosity; Mismatch repair; Mutation

Indexed keywords

CISPLATIN; GENE PRODUCT; PROTEIN MSH 2; UNCLASSIFIED DRUG;

ALLELE; ANIMAL CELL; APOPTOSIS; ARTICLE; BASE MISPAIRING; CARCINOGENESIS; CONTROLLED STUDY; DNA DAMAGE; DNA METHYLATION; GENE DOSAGE; GENE FREQUENCY; GENE LOCUS; GENE MUTATION; GENE REPRESSION; HETEROZYGOSITY; HOMOZYGOSITY; MOUSE; MUTATION RATE; NONHUMAN; PRIORITY JOURNAL;

ANIMALS; APOPTOSIS; BASE PAIR MISMATCH; CISPLATIN; DNA REPAIR; DNA-BINDING PROTEINS; GENE DOSAGE; GENOTYPE; HETEROZYGOTE; METHYLNITRONITROSOGUANIDINE; MICE; MICE, KNOCKOUT; MUTAGENESIS; MUTATION; MUTS HOMOLOG 2 PROTEIN; PROTO-ONCOGENE PROTEINS;

EID: 0035859002 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204449 Document Type: Article

Times cited : (19)

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