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Oncogene

Volumn 20, Issue 41, 2001, Pages 5826-5835

Cooperative and redundant effects of STAT5 and Ras signaling in BCR/ABL transformed hematopoietic cells

(4) Hoover, R R a Gerlach, M J a Koh, E Y a Daley, G Q a

a WHITEHEAD INSTITUTE FOR BIOMEDICAL RESEARCH (United States)

Author keywords

Apoptosis; BCR ABL; DNA damage; Ras; Signal transduction; STAT5

Indexed keywords

BCR ABL PROTEIN; PHOSPHATIDYLINOSITOL 3 KINASE; PROTEIN BCL X; RAS PROTEIN; STAT5 PROTEIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL PROLIFERATION; CELL SURVIVAL; CELL TRANSFORMATION; CONTROLLED STUDY; DNA DAMAGE; GAMMA RADIATION; HEMATOPOIETIC CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RADIOSENSITIVITY; SIGNAL TRANSDUCTION;

ANIMALS; APOPTOSIS; BCL-X PROTEIN; CELL DIVISION; CELL SURVIVAL; DNA DAMAGE; DNA-BINDING PROTEINS; FUSION PROTEINS, BCR-ABL; GENETIC VECTORS; HEMATOPOIETIC STEM CELLS; INTERLEUKIN-3; MILK PROTEINS; PHILADELPHIA CHROMOSOME; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; PROTO-ONCOGENE PROTEINS C-BCL-2; PROTO-ONCOGENE PROTEINS C-PIM-1; RAS PROTEINS; RETROVIRIDAE; SIGNAL TRANSDUCTION; STAT5 TRANSCRIPTION FACTOR; TRANS-ACTIVATORS; TRANSDUCTION, GENETIC;

ANIMALIA;

EID: 0035855648 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204549 Document Type: Article

Times cited : (77)

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