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Proceedings of the National Academy of Sciences of the United States of America

Volumn 98, Issue 6, 2001, Pages 3416-3421

The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation

(7) Forcet, Christelle a Ye, Xin b,c Granger, Laure a Corset, Véronique a Shin, Hwain b,c Bredesen, Dale E b,c Mehlen, Patrick a

a UNIVERSITÉ DE LYON (France)

b BUCK INSTITUTE FOR RESEARCH ON AGING (United States)

c UNIVERSITY OF CALIFORNIA (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; CASPASE 8; CASPASE 9; CELL SURFACE RECEPTOR; CYTOCHROME C; LIGAND; PROTEIN DCC; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; CELL DEATH; COLORECTAL CANCER; CONTROLLED STUDY; ENZYME ACTIVATION; GENE MUTATION; HUMAN; HUMAN CELL; IMMUNOBLOTTING; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; TUMOR SUPPRESSOR GENE;

APOPTOSIS; CASPASE 3; CASPASE 9; CASPASES; CELL ADHESION MOLECULES; CELL LINE, TRANSFORMED; COLORECTAL NEOPLASMS; ENZYME ACTIVATION; GENES, TUMOR SUPPRESSOR; HUMANS; RECEPTORS, CELL SURFACE; TUMOR CELLS, CULTURED; TUMOR SUPPRESSOR PROTEINS;

EID: 0035853158 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.051378298 Document Type: Article

Times cited : (182)

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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.