Hypoxia stimulates release of the soluble form of Fas ligand that inhibits endothelial cell apoptosis

Laboratory Investigation

Volumn 81, Issue 2, 2001, Pages 177-184

  Mogi, M ^a   Fukuo, K ^a   Yang, J ^a   Suhara, T ^a   Ogihara, T ^a  

^a OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; FAS ANTIGEN; METALLOPROTEINASE INHIBITOR; CARRIER PROTEIN; CFLAR PROTEIN, HUMAN; CFLAR PROTEIN, MOUSE; FAS LIGAND; FASL PROTEIN, MOUSE; FASLG PROTEIN, HUMAN; FLICE INHIBITORY PROTEIN; MEMBRANE PROTEIN; SIGNAL PEPTIDE;

APOPTOSIS; ARTICLE; CELL CULTURE; CONTROLLED STUDY; ENDOTHELIUM CELL; HUMAN; HUMAN CELL; HYPOXIA; OXYGEN TENSION; PRIORITY JOURNAL; PROTEIN SYNTHESIS; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; ANIMAL; CELL HYPOXIA; CYTOLOGY; GENE EXPRESSION REGULATION; GENETIC TRANSCRIPTION; GENETICS; MOUSE; PHYSIOLOGY; UMBILICAL VEIN; VASCULAR ENDOTHELIUM;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; CARRIER PROTEINS; CASP8 AND FADD-LIKE APOPTOSIS REGULATING PROTEIN; CELL HYPOXIA; CELLS, CULTURED; ENDOTHELIUM, VASCULAR; FAS LIGAND PROTEIN; GENE EXPRESSION REGULATION; HUMANS; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MEMBRANE GLYCOPROTEINS; MICE; PROTEIN BIOSYNTHESIS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; TRANSCRIPTION, GENETIC; UMBILICAL VEINS;

EID: 0035256309     PISSN: 00236837     EISSN: None     Source Type: Journal    
DOI: 10.1038/labinvest.3780225     Document Type: Article

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