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Volumn 50, Issue 4, 2001, Pages 803-809

Uncoupling protein 2: A possible link between fatty acid excess and impaired glucose-induced insulin secretion?

Author keywords

[No Author keywords available]

Indexed keywords

UNCOUPLING PROTEIN 2;

EID: 0035078830     PISSN: 00121797     EISSN: None     Source Type: Journal    
DOI: 10.2337/diabetes.50.4.803     Document Type: Article
Times cited : (214)

References (48)
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    • Long-term exposure of rat pancreatic islets to fatty acids inhibits glucose-induced insulin secretion and biosynthesis through a glucose fatty acid cycle
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    • Glucose-fatty acid cycle to inhibit glucose utilization and oxidation is not operative m fatty acid-cultured islets
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    • Influence of fatty acids on energy metabolism. II. Kinetics of changes in metabolic rates and changes in subcellular adenine nucleotide contents and Ph gradients following addition of octanoate and oleate in perfused rat liver
    • (1984) Eur J Biochem , vol.141 , pp. 231-236
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  • 43
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    • Feasibility of a mitochondrial pyruvate malate shuttle in pancreatic islets: Further implication of cytosolic NADPH in insulin secretion
    • (1995) J Biol Chem , vol.270 , pp. 20051-20058
    • MacDonald, M.J.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.