Are genetically modified mice useful for the understanding of acute pancreatitis?

FASEB Journal

Volumn 15, Issue 6, 2001, Pages 893-897

  Pastor, Catherine M ^a,b   Frossard, Jean Louis ^b  

^a UNIVERSITY OF GENEVA   (Switzerland)

^b Division d'Hépatologie et de Gastroentérologie   (Switzerland)

Author keywords

Acute pancreatitis; IL 1; Knockout mice; Metallothionein; NK1R; Tumor necrosis factor

Indexed keywords

CHEMOKINE; CYTOKINE; INTERLEUKIN 1; LIGAND; RECEPTOR; TUMOR NECROSIS FACTOR;

ACUTE PANCREATITIS; ANIMAL MODEL; CYTOKINE PRODUCTION; CYTOKINE RELEASE; DNA MODIFICATION; GENE DELETION; IMMUNE SYSTEM; INFLAMMATION; KNOCKOUT MOUSE; MOUSE; NONHUMAN; PRIORITY JOURNAL; REVIEW; SYSTEMIC CIRCULATION;

ACUTE DISEASE; ANIMALS; CATHEPSIN B; CYTOKINES; DISEASE MODELS, ANIMAL; INTERCELLULAR ADHESION MOLECULE-1; METALLOTHIONEIN; MICE; MICE, KNOCKOUT; PANCREATITIS; RECEPTORS, CHEMOKINE; RECEPTORS, NEUROKININ-1; SUPEROXIDE DISMUTASE;

ANIMALIA;

EID: 0035066833     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.00-0672rev     Document Type: Review

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