Defining a common region of deletion at 13q21 in human cancers

Genes Chromosomes and Cancer

Volumn 31, Issue 4, 2001, Pages 333-344

  Chen, Ceshi ^a   Brabham, Will W ^a   Stultz, Brian G ^b   Frierson Jr, Henry F ^a   Carl Barrett, J ^c   Sawyers, Charles L ^d   Isaacs, John T ^e   Dong, Jin Tang ^a,b  

^a UNIVERSITY OF VIRGINIA HEALTH SYSTEM   (United States)

^b UNIVERSITY OF VIRGINIA   (United States)

^c NATIONAL CANCER INSTITUTE   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

^e JOHNS HOPKINS UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CONTIG;

ARTICLE; BACTERIAL ARTIFICIAL CHROMOSOME; BREAST CANCER; CHROMOSOME 13Q; CHROMOSOME DELETION 13; CHROMOSOME MAP; CONTROLLED STUDY; ENDOMETRIUM CANCER; EXPRESSED SEQUENCE TAG; GENE EXPRESSION; GENE MUTATION; GENE SEQUENCE; GENETIC ANALYSIS; HETEROZYGOSITY LOSS; HOMOZYGOSITY; HUMAN; HUMAN CELL; NUCLEOTIDE SEQUENCE; OVARY CANCER; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; PROSTATE CANCER; SEQUENCE TAGGED SITE; SINGLE STRAND CONFORMATION POLYMORPHISM; TUMOR SUPPRESSOR GENE; UROGENITAL TRACT CANCER; UTERINE CERVIX CANCER;

CHROMOSOME DELETION; CHROMOSOMES, ARTIFICIAL, BACTERIAL; CHROMOSOMES, ARTIFICIAL, YEAST; CHROMOSOMES, HUMAN, PAIR 13; DNA-BINDING PROTEINS; GENES, TUMOR SUPPRESSOR; HOMOZYGOTE; HUMANS; KRUPPEL-LIKE TRANSCRIPTION FACTORS; LOSS OF HETEROZYGOSITY; MOLECULAR SEQUENCE DATA; NEOPLASMS; PHYSICAL CHROMOSOME MAPPING; REPRESSOR PROTEINS; SEQUENCE TAGGED SITES; TRANSCRIPTION FACTORS; TUMOR CELLS, CULTURED;

BACTERIA (MICROORGANISMS);

EID: 0034932249     PISSN: 10452257     EISSN: None     Source Type: Journal    
DOI: 10.1002/gcc.1152     Document Type: Article

References (36)

1. Overrepresentation of 3q and 8q material and loss of 18q material are recurrent findings in advanced human ovarian cancer
2. The developmentally regulated transcription factor AP-2 is involved in c-erbB-2 over-expression in human mammary carcinoma
3. AP-2: A nuclear effector of malignant transformation by ras oncogene
4. High resolution YAC-cosmid-STS map of human chromosome 13
5. Genetic alterations in untreated metastases and androgen-independent prostate cancer detected by comparative genomic hybridization and allelotyping
6. Deletion at 13q21 is associated with aggressive prostate cancers
7. Localization of multiple melanoma tumor-suppressor genes on chromosome 11 by use of homozygosity mapping-of-deletions analysis
8. An STS-based map of the human genome
9. Three distinct regions of allelic loss at 13q14, 13q21-22, and 13q33 in prostate cancer
10. Transcriptional regulation of the AP-2alpha promoter by BTEB-1 and AP-2rep, a novel wt-1/egr-related zinc finger repressor
11. Somatic deletions in hereditary breast cancers implicate 13q21 as a putative novel breast cancer susceptibility locus
12. Identification of gains and losses of DNA sequences in primary bladder cancer by comparative genomic hybridization
13. Chromosomal abnormalities in glioblastoma multiforme tumors and glioma cell lines detected by comparative genomic hybridization
14. DNA copy number losses in human neoplasms
15. Localization of the human homologue of the Drosophila dachshund gene (DACH) to chromosome 13q21
16. SSCP analysis of long DNA fragments in low pH gel
17. Comparative genomic hybridization of malignant fibrous histiocytoma reveals a novel prognostic marker
18. Comparative genomic hybridization reveals complex genetic changes in primary breast cancer tumors and their cell lines
19. Expression of activator protein 2 in prostate cancer is related to tumor differentiation and cell proliferation
20. Loss of chromosome 13 is the most frequent genomic imbalance in malignant fibrous histiocytomas. A comparative genomic hybridization analysis of a series of 30 cases
21. Methylation of the 5′ CpG island of the endothelin B receptor gene is common in human prostate cancer
22. A frameshift mutation of the endothelin B receptor gene (EDNRB) in the prostate cancer cell line LNCaP
23. Investigation of genetic alterations associated with the grade of astrocytic tumor by comparative genomic hybridization
24. Genetic alterations in prostate cancer cell lines detected by comparative genomic hybridization
25. Genetic alterations in hormone-refractory recurrent prostate carcinomas
26. Comparative genomic hybridization reveals novel chromosome deletions in 90 primary soft tissue tumors
27. Flow cytometric and karyotypic analysis of a primary small cell carcinoma of the prostate: A xenografted cell line
28. Genomic structure and DNA binding properties of the human zinc finger transcriptional repressor AP-2rep (KLF12)
29. A gene map of the human genome
30. Characterization of two novel protocadherins (PCDH8 and PCDH9) localized on human chromosome 13 and mouse chromosome 14
31. Gains and losses of DNA sequences in liposarcomas evaluated by comparative genomic hybridization
32. Genetic imbalances in 67 synovial sarcomas evaluated by comparative genomic hybridization
33. Genetic changes in primary and recurrent prostate cancer by comparative genomic hybridization
34. Comparative genomic hybridization analysis of chromosomal changes occurring during development of acquired resistance to cisplatin in human ovarian carcinoma cells
35. Inactivation of the tumor suppressor PTEN/MMAC1 in advanced human prostate cancer through loss of expression