메뉴 건너뛰기




Volumn 278, Issue 3, 2000, Pages 550-556

Anisomycin affects both pro- and antiapoptotic mechanisms in PC12 cells

Author keywords

Akt; Anisomycin; Apoptosis; Bcl 2; Gene expression; Mitogen activated protein kinases; Nerve growth factor; Neuronal differentiation; PC12 cells

Indexed keywords

ANISOMYCIN; GENE PRODUCT; MITOGEN ACTIVATED PROTEIN KINASE; NERVE GROWTH FACTOR; PROTEIN BCL 2; PROTEIN KINASE B; UNCLASSIFIED DRUG;

EID: 0034736116     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.2000.3836     Document Type: Article
Times cited : (34)

References (42)
  • 9
    • 0021359709 scopus 로고
    • The importance of both early and delayed responses in the biological actions of nerve growth factor
    • (1984) Trends Neurosci. , vol.7 , pp. 91-94
    • Greene, L.A.1
  • 24
    • 0034050833 scopus 로고    scopus 로고
    • Anisomycin uses multiple mechanisms to stimulate mitogen-activated protein kinases and gene expression and to inhibit neuronal differentiation in PC12 pheochromocytoma cells
    • (2000) Eur. J. Neurosci. , vol.12 , pp. 527-532
    • Torocsik, B.1    Szeberenyi, J.2
  • 25
    • 0028963084 scopus 로고
    • Requirement for phosphatidylinositol 3-kinase in the prevention of apoptosis by nerve growth factor
    • (1995) Science , vol.267 , pp. 2003-2006
    • Yao, R.1    Cooper, G.M.2
  • 35
    • 0033517244 scopus 로고    scopus 로고
    • Akt is more than just a Bad kinase
    • (1999) Nature , vol.401 , pp. 33-34
    • Khwaja, A.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.