Bcl-2 does not inhibit the permeability transition pore in mouse liver mitochondria

Toxicology

Volumn 151, Issue 1-3, 2000, Pages 65-72

  Yang, Joy C ^a   Kahn, Axel ^b   Cortopassi, Gino ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b INSTITUT COCHIN   (France)

Author keywords

Apoptosis; Caspase; Fas ligand; Mitochondria; Permeability transition pore

Indexed keywords

ATRACTYLOSIDE; CALCIUM ION; CASPASE 3; CYCLOSPORIN A; CYTOCHROME C; FAS ANTIBODY; FAS ANTIGEN; PROTEIN BCL 2; TERT BUTYL HYDROPEROXIDE; UNCLASSIFIED DRUG;

ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; ENZYME BLOOD LEVEL; ENZYME RELEASE; HYPOTHESIS; LIVER MITOCHONDRION; MALE; MEMBRANE PERMEABILITY; MITOCHONDRION SWELLING; MORPHOLOGY; MOUSE; NONHUMAN; PERMEABILITY TRANSITION PORE; POROSITY; PRIORITY JOURNAL; PROTECTION; PROTEIN EXPRESSION; TRANSGENIC MOUSE; WESTERN BLOTTING;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; BLOTTING, WESTERN; CYTOCHROME C GROUP; HUMANS; LIGHT; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MITOCHONDRIA, LIVER; MITOCHONDRIAL SWELLING; PERMEABILITY; PROTO-ONCOGENE PROTEINS C-BCL-2; SCATTERING, RADIATION;

MUS MUSCULUS; RODENTIA;

EID: 0034718949     PISSN: 0300483X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0300-483X(00)00298-5     Document Type: Article

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