A FE65 polymorphism associated with risk of developing sporadic late-onset Alzheimer's disease but not with Aβ loading in brains

Neuroscience Letters

Volumn 293, Issue 1, 2000, Pages 29-32

  Lambert, Jean Charles ^a,b   Mann, David ^c   Goumidi, Louisa ^a   Harris, Judith ^b   Pasquier, Florence ^d   Frigard, Bernard ^e   Cottel, Dominique ^a   Lendon, Corinne ^b   Iwatsubo, Takeshi ^f   Amouyel, Philippe ^a   Chartier Harlin, Marie Christine ^a  

^a INSTITUT PASTEUR DE LILLE   (France)

^b Molecular Psychiatry Department   (United Kingdom)

^c UNIVERSITY OF MANCHESTER   (United Kingdom)

^d UNIV LILLE   (France)

^e Ctr Geriartrie de Wasquehal Molinel   (France)

^f University of   (Japan)

Author keywords

A peptide; Alzheimer; Brain; FE65; Polymorphism

Indexed keywords

AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; PROTEIN; PROTEIN FE65; UNCLASSIFIED DRUG;

AGED; ALZHEIMER DISEASE; ARTICLE; AUTOPSY; CONTROLLED STUDY; EXON; FEMALE; GENE; GENE DELETION; GENETIC POLYMORPHISM; GENOTYPE; HUMAN; HUMAN TISSUE; INTRON; MAJOR CLINICAL STUDY; MALE; ONSET AGE; PRIORITY JOURNAL; PROTEIN DOMAIN; RNA SPLICING;

AGE OF ONSET; AGED; AGED, 80 AND OVER; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; BRAIN; FEMALE; GENE DELETION; GENETIC PREDISPOSITION TO DISEASE; HUMAN; MALE; NERVE TISSUE PROTEINS; NUCLEAR PROTEINS; REFERENCE VALUES; SUPPORT, NON-U.S. GOV'T;

EID: 0034693448     PISSN: 03043940     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0304-3940(00)01477-4     Document Type: Article

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