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Volumn 96, Issue 12, 2000, Pages 3915-3921

Bcl-2 expression restores the leukemogenic potential of a BCR/ABL mutant defective in transformation

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN BAD; PROTEIN BCL 2;

EID: 0034548831     PISSN: 00064971     EISSN: None     Source Type: Journal    
DOI: 10.1182/blood.v96.12.3915.h8003915_3915_3921     Document Type: Article
Times cited : (29)

References (45)
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  • 22
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    • BCR/abl leads to the constitutive activation of Stat proteins, and shares an epitope with tyrosine phosphorylated Stats
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    • Frank, D.A.1    Varticovski, L.2
  • 23
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    • P210 and P190(BCR/ABL) induce the tyrosine phosphorylation and DNA binding activity of multiple specific STAT family members
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    • Ilaria R.L., Jr.1    Van Etten, R.A.2
  • 25
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    • Signal transducer and activator of transcription (STAT)5 activation by BCR/ABL is dependent on intact Src homology (SH)3 and SH2 domains of BCR/ABL and is required for leukemogenesis
    • (1999) J Exp Med , vol.189 , pp. 1229-1242
    • Nieborowska-Skorska, M.1    Wasik, M.A.2    Slupianek, A.3
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    • Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery
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  • 42
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    • High expression of bcl-2 protein in acute myeloid leukemia cells is associated with poor response to chemotherapy
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    • Campos, L.1    Rouault, J.P.2    Sabido, O.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.