Transgenic rat hearts expressing a human cardiac troponin T deletion reveal diastolic dysfunction and ventricular arrhythmias

Cardiovascular Research

Volumn 47, Issue 2, 2000, Pages 254-264

  Frey, Norbert ^a,b   Franz, Wolfgang M ^a   Gloeckner, Katharina ^a   Degenhardt, Michael ^a   Müller, Matthias ^a   Müller, Oliver ^a   Merz, Hartmut ^a   Katus, Hugo A ^a  

^a UNIVERSITY OF LÜBECK   (Germany)

^b UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

Cardiomyopathy; Contractile function; Hypertrophy; Ventricular arrhythmias

Indexed keywords

MUTANT PROTEIN; TROPONIN T;

ANIMAL CELL; ARRHYTHMOGENESIS; ARTICLE; CONTROLLED STUDY; DIASTOLE; EXERCISE; FAMILIAL DISEASE; GENE DELETION; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART VENTRICLE ARRHYTHMIA; HISTOLOGY; HUMAN; HYPERTROPHIC CARDIOMYOPATHY; IMMUNOHISTOCHEMISTRY; NEWBORN; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; RAT; SYSTOLE; TRANSGENIC ANIMAL;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; BLOTTING, WESTERN; CARDIOMEGALY; CARDIOMYOPATHY, HYPERTROPHIC; COS CELLS; DIASTOLE; GENE DELETION; GENE EXPRESSION; HUMANS; MALE; MYOCARDIUM; PERFUSION; RATS; RATS, SPRAGUE-DAWLEY; SARCOMERES; SYSTOLE; TROPONIN T; VENTRICULAR DYSFUNCTION;

EID: 0033886318     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0008-6363(00)00114-0     Document Type: Article

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