A truncated cardiac troponin T molecule in transgenic mice suggests multiple cellular mechanisms for familial hypertrophic cardiomyopathy

Journal of Clinical Investigation

Volumn 101, Issue 12, 1998, Pages 2800-2811

  Tardiff, Jil C ^a,d   Factor, Stephen M ^b   Tompkins, Brian D ^d   Hewett, Timothy E ^c   Palmer, Bradley M ^d   Moore, Russell L ^d   Schwartz, Steve ^b   Robbins, Jeffrey ^b   Leinwand, Leslie A ^d  

^a Columbia University ^*  (United States)

^b ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

^c CINCINNATI CHILDREN S HOSPITAL RESEARCH FOUNDATION   (United States)

^d UNIVERSITY OF COLORADO   (United States)

Author keywords

Cardiomyopathy; Mice; Myofibrils; Physiopathology; Troponin

Indexed keywords

MYOSIN HEAVY CHAIN; TROPONIN T;

ALLELE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; GENE MUTATION; HEART VENTRICLE HYPERTROPHY; HYPERTROPHIC CARDIOMYOPATHY; ISOLATED HEART; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SARCOMERE; SUDDEN DEATH; SURVIVAL RATE; TRANSGENE; TRANSGENIC MOUSE;

EID: 0032526155     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI2389     Document Type: Article

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