Loss of p19(ARF) eliminates the requirement for the pRB-binding motif in simian virus 40 large T antigen-mediated transformation

Molecular and Cellular Biology

Volumn 20, Issue 20, 2000, Pages 7624-7633

  Chao, H H A ^a   Buchmann, A M ^a   DeCaprio, J A ^a  

^a DANA FARBER CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

HEAT SHOCK PROTEIN 40; PROTEIN P16; PROTEIN P19 ALTERNATE READING FRAME; PROTEIN P53; RETINOBLASTOMA PROTEIN; TRANSCRIPTION FACTOR; UNCLASSIFIED DRUG; VIRUS LARGE T ANTIGEN;

ANIMAL CELL; ANTIGEN BINDING; ARTICLE; CELL TRANSFORMATION; EMBRYO; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DEPLETION; PROTEIN DOMAIN; PROTEIN FAMILY; SIMIAN VIRUS 40;

AMINO ACID MOTIFS; ANIMAL; ANTIGENS, VIRAL, TUMOR; BLOTTING, WESTERN; CARRIER PROTEINS; CELL COUNT; CELL DIVISION; CELL LINE, TRANSFORMED; CELL TRANSFORMATION, NEOPLASTIC; CONTACT INHIBITION; FIBROBLASTS; GENE DELETION; GENE EXPRESSION REGULATION, NEOPLASTIC; GENES, REPORTER; MICE; P14ARF PROTEIN; PROMOTER REGIONS (GENETICS); PROTEIN BINDING; PROTEIN P16; PROTEIN P53; PROTEINS; RETINOBLASTOMA PROTEIN; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TRANSCRIPTION FACTORS;

EID: 0033800987     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.20.20.7624-7633.2000     Document Type: Article

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