The stress-responsive MAP kinase p38 is activated by low-flow ischemia in the in situ porcine heart

Journal of Molecular and Cellular Cardiology

Volumn 32, Issue 10, 2000, Pages 1787-1794

  Lüss, Hartmut ^a   Neumann, Joachim ^a   Schmitz, Wilhelm ^a   Schulz, Rainer ^b   Heusch, Gerd ^b  

^a UNIVERSITY OF MÜNSTER   (Germany)

^b UNIVERSITY HOSPITAL ESSEN   (Germany)

Author keywords

Calsequestrin; Hibernation; Phospholamban; Reperfusion; SERCA2a; Stunning; Troponin inhibitor

Indexed keywords

CALSEQUESTRIN; CREATINE PHOSPHATE; MITOGEN ACTIVATED PROTEIN KINASE; PHOSPHOLAMBAN; STRESS ACTIVATED PROTEIN KINASE; TROPONIN I;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; CORONARY ARTERY BLOOD FLOW; ENZYME ACTIVATION; HEART MUSCLE BIOPSY; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; MYOCARDIAL HIBERNATION; NONHUMAN; PHOSPHORYLATION; PRIORITY JOURNAL; PROTEIN EXPRESSION; STUNNED HEART MUSCLE; SWINE;

ANIMALIA; SUIDAE; SUS SCROFA;

EID: 0033736044     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1006/jmcc.2000.1213     Document Type: Article

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